Institution
Shandong University
Education•Jinan, Shandong, China•
About: Shandong University is a education organization based out in Jinan, Shandong, China. It is known for research contribution in the topics: Laser & Cancer. The organization has 99070 authors who have published 99160 publications receiving 1625094 citations. The organization is also known as: Shāndōng Dàxué.
Topics: Laser, Cancer, Apoptosis, Microstructure, Cell growth
Papers published on a yearly basis
Papers
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TL;DR: The results suggest that this garlic skin-derived 3D hierarchical porous carbon is a promising electrode material for high-performance supercapacitors.
Abstract: A three-dimensional hierarchical porous carbon is synthesized via a facile chemical activation route with garlic skin as the precursor and KOH as the activating agent. The as-obtained carbon presents a high specific surface area of 2818 m2 g−1 and a hierarchical porous architecture containing macroporous frameworks, mesopores (2–4 nm), and micropores (0.6–1.0 nm). As the electrode material for a supercapacitor, due to its unique interconnected porous structure, this garlic skin-derived carbon exhibits excellent electrochemical performance and cycling stability. At a current density of 0.5 A g−1, the capacitance is up to 427 F g−1 (162 F cm−3). Even at a high current density of 50 A g−1, the capacitance can be maintained to a high value of 315 F g−1 (120 F cm−3). After charging–discharging at a current density of 4.5 A g−1 for 5000 cycles, the capacitance retention is as high as 94%. The results suggest that this garlic skin-derived 3D hierarchical porous carbon is a promising electrode material for high-performance supercapacitors.
331 citations
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Shanghai Jiao Tong University1, University of Science and Technology of China2, Nanjing University3, Peking University4, Central South University5, Second Military Medical University6, Shandong University7, Tianjin Medical University8, Harbin Medical University9, China Medical University (PRC)10, Chongqing Medical University11, Fudan University12, Sichuan University13, Peking Union Medical College Hospital14, Fujian Medical University15
TL;DR: The main contents include epidemiology of type 2 diabetes in China; diagnosis and classification of diabetes; primary, secondary, and tertiary diabetes prevention; diabetes education and management support; blood glucose monitoring; integrated control targets for type 1 diabetes and treatments for hyperglycaemia.
Abstract: The prevalence of diabetes in China has increased rapidly from 0.67% in 1980 to 10.4% in 2013, with the aging of the population and westernization of lifestyle. Since its foundation in 1991, the Chinese Diabetes Society (CDS) has been dedicated to improving academic exchange and the academic level of diabetes research in China. From 2003 to 2014, four versions of Chinese diabetes care guidelines have been published. The guidelines have played an important role in standardizing clinical practice and improving the status quo of diabetes prevention and control in China. Since September 2016, the CDS has invited experts in cardiovascular diseases, psychiatric diseases, nutrition, and traditional Chinese medicine to work with endocrinologists from the CDS to review the new clinical research evidence related to diabetes over the previous 4 years. Over a year of careful revision, this has resulted in the present, new version of guidelines for prevention and care of type 2 diabetes in China. The main contents include epidemiology of type 2 diabetes in China; diagnosis and classification of diabetes; primary, secondary, and tertiary diabetes prevention; diabetes education and management support; blood glucose monitoring; integrated control targets for type 2 diabetes and treatments for hyperglycaemia; medical nutrition therapy; exercise therapy for type 2 diabetes; smoking cessation; pharmacologic therapy for hyperglycaemia; metabolic surgery for type 2 diabetes; prevention and treatment of cardiovascular and cerebrovascular diseases in patients with type 2 diabetes; hypoglycaemia; chronic diabetic complications; special types of diabetes; metabolic syndrome; and diabetes and traditional Chinese medicine.
331 citations
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TL;DR: In this article, a comprehensive literature review is needed because some related concepts are not clear and the precision of models still need to be promoted in this field, and conclusions are drawn for the future study in two major points: 1) the accuracy of current energy consumption models could be improved through introducing the correlation analysis of machine tools, parts, tools and processing condition.
331 citations
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TL;DR: In this article, a search for new resonant and non-resonant high-mass phenomena in dielectron and dimuon fi nal states was conducted using 36 : 1 fb(-1) of proton-proton collision data.
Abstract: A search is conducted for new resonant and non-resonant high-mass phenomena in dielectron and dimuon fi nal states. The search uses 36 : 1 fb(-1) of proton-proton collision data, collected at root ...
329 citations
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TL;DR: It is found that NLRP3 deficiency ameliorated cerebral injury in mice after ischemic stroke by reducing infarcts and blood–brain barrier (BBB) damage and that NOX2 deficiency improved outcomes after ischemia stroke by mediatingNLRP3 signaling.
Abstract: Although the innate immune response to induce postischemic inflammation is considered as an essential step in the progression of cerebral ischemia injury, the role of innate immunity mediator NLRP3 in the pathogenesis of ischemic stroke is unknown. In this study, focal ischemia was induced by middle cerebral artery occlusion in NLRP3−/−, NOX2−/−, or wild-type (WT) mice. By magnetic resonance imaging (MRI), Evans blue permeability, and electron microscopic analyses, we found that NLRP3 deficiency ameliorated cerebral injury in mice after ischemic stroke by reducing infarcts and blood–brain barrier (BBB) damage. We further showed that the contribution of NLRP3 to neurovascular damage was associated with an autocrine/paracrine pattern of NLRP3-mediated interleukin-1β (IL-1β) release as evidenced by increased brain microvessel endothelial cell permeability and microglia-mediated neurotoxicity. Finally, we found that NOX2 deficiency improved outcomes after ischemic stroke by mediating NLRP3 signaling. This study for the first time shows the contribution of NLRP3 to neurovascular damage and provides direct evidence that NLRP3 as an important target molecule links NOX2-mediated oxidative stress to neurovascular damage in ischemic stroke. Pharmacological targeting of NLRP3-mediated inflammatory response at multiple levels may help design a new approach to develop therapeutic strategies for prevention of deterioration of cerebral function and for the treatment of stroke.
329 citations
Authors
Showing all 99666 results
Name | H-index | Papers | Citations |
---|---|---|---|
Jing Wang | 184 | 4046 | 202769 |
Yang Gao | 168 | 2047 | 146301 |
Gang Chen | 167 | 3372 | 149819 |
Yang Yang | 164 | 2704 | 144071 |
Andrew D. Hamilton | 151 | 1334 | 105439 |
Ben Zhong Tang | 149 | 2007 | 116294 |
Yoshio Bando | 147 | 1234 | 80883 |
Guanrong Chen | 141 | 1652 | 92218 |
Karl Jakobs | 138 | 1379 | 97670 |
Jun Chen | 136 | 1856 | 77368 |
Shu Li | 136 | 1001 | 78390 |
Hui Li | 135 | 2982 | 105903 |
Lei Zhang | 135 | 2240 | 99365 |
Elizaveta Shabalina | 133 | 1421 | 92273 |
George A. Calin | 133 | 654 | 106942 |