C
Christer Hogstrand
Researcher at King's College London
Publications - 347
Citations - 14664
Christer Hogstrand is an academic researcher from King's College London. The author has contributed to research in topics: Zinc & Metallothionein. The author has an hindex of 61, co-authored 323 publications receiving 11846 citations. Previous affiliations of Christer Hogstrand include European Food Safety Authority & University of Kentucky.
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Effect and mechanism of waterborne prolonged Zn exposure influencing hepatic lipid metabolism in javelin goby Synechogobius hasta
Chao Huang,Zhi Luo,Zhi Luo,Christer Hogstrand,Feng Chen,Xi Shi,Qi-Liang Chen,Yu-Feng Song,Ya-Xiong Pan +8 more
TL;DR: Evidence is provided that chronic Zn exposure differentially influenced mRNA expression and activities of genes and enzymes involved in lipogenic and lipolytic metabolism in a duration‐dependent manner, and new insight is provided into the relationship between metal elements and lipid metabolism.
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Expression of the ZIP/SLC39A transporters in β-cells: a systematic review and integration of multiple datasets.
TL;DR: ZIP6 and ZIP7 are proposed to be key functional orthologues in human and rodent β-cells and highlight these zinc importers as important targets for exploring associations between zinc status and normal physiology of β- cells and their decline in Type 2 Diabetes.
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Scientific Opinion on the safety and efficacy of concentrated liquid L-lysine (base), concentrated liquid L-lysine monohydrochloride and L-lysine monohydrochloride produced by Escherichia coli (FERM BP-10941) for all animal species, based on three dossiers submitted by Ajinomoto Eurolysine SAS
Gabriele Aquilina,Vasileios Bampidis,Maria de Lourdes Bastos,Lucio G. Costa,G. Flachowsky,Mikołaj Antoni Gralak,Christer Hogstrand,Lubomir Leng,S. López Puente,Giovanna Martelli,Baltasar Mayo,Fernando Ramos,Derek Renshaw,Guido Rychen,Maria Saarela,K. Sejrsen,P. Van Beelen,Robert John Wallace,Johannes Westendorf +18 more
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Prolonged stimulation of insulin release from MIN6 cells causes zinc depletion and loss of β-cell markers.
TL;DR: The data suggest hyperglycaemia-induced zinc depletion may contribute to loss of β-cell markers and promote β- cell dedifferentiation through disrupting expression of key transcription factors.