C
Christer Hogstrand
Researcher at King's College London
Publications - 347
Citations - 14664
Christer Hogstrand is an academic researcher from King's College London. The author has contributed to research in topics: Zinc & Metallothionein. The author has an hindex of 61, co-authored 323 publications receiving 11846 citations. Previous affiliations of Christer Hogstrand include European Food Safety Authority & University of Kentucky.
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Journal ArticleDOI
Naturally high levels of zinc and metallothionein in liver of several species of the squirrelfish family from Queensland, Australia
Christer Hogstrand,Carl Haux +1 more
TL;DR: The results indicate that the high concentrations of MT and Zn in Holocentridae liver are linked to normal physiological processes and suggest that the Holocent Ridae family may be a unique model for future studies of Zn metabolism and the function of MT.
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Protein kinase CK2 opens the gate for zinc signaling.
TL;DR: The importance of zinc in biology is evidenced by the growing number of disease states that directly involve aberrant levels of zinc, including neurodegeneration, inflammation, diabetes, cancer and more.
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Machine Learning for Environmental Toxicology: A Call for Integration and Innovation.
Thomas H. Miller,Matteo Gallidabino,James I. MacRae,Christer Hogstrand,Nicolas R. Bury,Leon Barron,Jason Snape,Stewart F. Owen +7 more
TL;DR: Machine learning is an example of a disruptive research technology, which is urgently needed to cope with the complexity and scale of work required within environmental toxicology.
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Identification, cloning and characterization of a plasma membrane zinc efflux transporter, TrZnT-1, from fugu pufferfish (Takifugu rubripes)
TL;DR: An orthologue of the mammalian ZnT-1 (zinc transporter-1) gene was cloned from the intestine of the torafugu pufferfish (Takifugu rubripes), demonstrating that this gene predates the evolution of land-living vertebrates.
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FXR-mediated inhibition of autophagy contributes to FA-induced TG accumulation and accordingly reduces FA-induced lipotoxicity
TL;DR: This study indicated that farnesoid X receptor-cyclic AMP-responsive element-binding protein (CREB) axis was the pivotal physiological switch regulatingFA-induced changes of autophagy and lipid metabolism, which represented cellular defenses against FA-induced lipotoxicity.