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Israel Liberzon

Researcher at University of Michigan

Publications -  298
Citations -  29883

Israel Liberzon is an academic researcher from University of Michigan. The author has contributed to research in topics: Prefrontal cortex & Amygdala. The author has an hindex of 80, co-authored 280 publications receiving 26492 citations. Previous affiliations of Israel Liberzon include University of Illinois at Chicago & Mental Health Services.

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General distress is more important than PTSD's cognition and mood alterations factor in accounting for PTSD and depression's comorbidity.

TL;DR: The present study suggests that the high rate of comorbidity between posttraumatic stress disorder (PTSD) and major depressive disorder is more related to underlying general distress or negative affectivity than the symptom categories of the PTSD diagnostic criteria.
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Genomic influences on self-reported childhood maltreatment

Shareefa Dalvie, +89 more
TL;DR: This is the first large-scale genetic study to identify specific variants associated with self-reported childhood maltreatment and quantify genetic overlap of childhood malt treatment with mental and physical health-related phenotypes, and condition the top hits from the authors' analyses when such overlap is present.
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Does neuroimaging research examining the pathophysiology of posttraumatic stress disorder require medication-free patients?

TL;DR: The empirical evidence for relevant medications having a confounding effect on task performance or CBF in relevant areas remains sparse for most psychotropic medications among patients with PTSD, but considerable evidence is accumulating for 2 of the most commonly prescribed medication classes in healthy controls.
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Attentional processes in posttraumatic stress disorder and the associated changes in neural functioning

TL;DR: Using instruments that allow assessment of behavioral and neurophysiological attention components will be necessary to understand attention deficits in PTSD and directions for future research are offered to clarify some of the mixed findings.
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Trauma exposure and sleep: using a rodent model to understand sleep function in PTSD.

TL;DR: A model which implicates abnormal activity in the locus coeruleus (LC), an important modifier of sleep–wake regulation, as the source of sleep abnormalities and memory abnormalities seen in PTSD is proposed.