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Lakshmi Pillai

Researcher at University of Texas Medical Branch

Publications -  44
Citations -  1517

Lakshmi Pillai is an academic researcher from University of Texas Medical Branch. The author has contributed to research in topics: Internal medicine & Flavocoxid. The author has an hindex of 19, co-authored 36 publications receiving 1409 citations. Previous affiliations of Lakshmi Pillai include University of Kentucky & University at Buffalo.

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Gene ontology annotations and resources

Judith A. Blake, +134 more
TL;DR: The Gene Ontology (GO) Consortium is a community-based bioinformatics resource that classifies gene product function through the use of structured, controlled vocabularies and has been expanded not only to cover new areas of biology through focused interaction with experts, but also to capture greater specificity in all areas of the ontology.
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The Gene Ontology: Enhancements for 2011

Judith A. Blake, +146 more
TL;DR: The Gene Ontology (GO) is a community bioinformatics resource that represents gene product function through the use of structured, controlled vocabularies and continues to expand and improve as a result of targeted ontology development.
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The type III secretion system and cytotoxic enterotoxin alter the virulence of Aeromonas hydrophila.

TL;DR: This is the first report describing a correlation between the TTSS and Act of A. hydrophila and the production of lactones, and the effects of act and aopB gene deletions on lactone production could be complemented with the native genes.
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DNA Adenine Methyltransferase Influences the Virulence of Aeromonas hydrophila

TL;DR: The dam gene was essential for the viability of the bacterium, and overproduction of Dam in A. hydrophila SSU, using an arabinose-inducible, PBAD promoter-based system, reduced the virulence of this pathogen.
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Identification of Aeromonas hydrophila Cytotoxic Enterotoxin-induced Genes in Macrophages Using Microarrays

TL;DR: The array data provided for the first time a global view of Act-mediated signal transduction and clearly demonstrated an inflammatory response and apoptosis mediated by this toxin in host cells at the molecular level.