M
Merlin L. Robb
Researcher at Walter Reed Army Institute of Research
Publications - 314
Citations - 21296
Merlin L. Robb is an academic researcher from Walter Reed Army Institute of Research. The author has contributed to research in topics: Viral load & Vaccination. The author has an hindex of 61, co-authored 279 publications receiving 17649 citations. Previous affiliations of Merlin L. Robb include Henry M. Jackson Foundation for the Advancement of Military Medicine & United States Military HIV Research Program.
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Journal ArticleDOI
Vaccination with ALVAC and AIDSVAX to Prevent HIV-1 Infection in Thailand
Supachai Rerks-Ngarm,Punnee Pitisuttithum,Sorachai Nitayaphan,Jaranit Kaewkungwal,Joseph Chiu,Robert Paris,Nakorn Premsri,Chawetsan Namwat,Mark de Souza,Elizabeth Adams,Michael Benenson,Sanjay Gurunathan,Jim Tartaglia,John G. McNeil,Donald P. Francis,Donald Stablein,Deborah L. Birx,Supamit Chunsuttiwat,Chirasak Khamboonruang,Prasert Thongcharoen,Merlin L. Robb,Nelson L. Michael,Prayura Kunasol,Jerome H. Kim +23 more
TL;DR: This ALVAC-HIV and AIDSVAX B/E vaccine regimen may reduce the risk of HIV infection in a community-based population with largely heterosexual risk and offer insight for future research.
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Safety and Efficacy of Single-Dose Ad26.COV2.S Vaccine against Covid-19.
Jerald C. Sadoff,Glenda Gray,An Vandebosch,Vicky Cardenas,Georgi Shukarev,Beatriz Grinsztejn,Paul A. Goepfert,Carla Truyers,Hein Fennema,Bart Spiessens,Kim Offergeld,Gert Scheper,Kimberly L Taylor,Merlin L. Robb,John J. Treanor,Dan H. Barouch,Jeffrey J. Stoddard,Martin Ryser,Mary A. Marovich,Kathleen M. Neuzil,Lawrence Corey,Nancy Cauwenberghs,Tamzin Tanner,Karin Hardt,Javier Ruiz-Guiñazú,Mathieu Le Gars,Hanneke Schuitemaker,Johan Van Hoof,Frank Struyf,Macaya Douoguih +29 more
TL;DR: A single dose of Ad26.COV2.S protected against symptomatic Covid-19 and asymptomatic SARS-CoV-2 infection and was effective against severe–critical disease, including hospitalization and death, in an international, randomized, double-blind, placebo-controlled, phase 3 trial.
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Immune-Correlates Analysis of an HIV-1 Vaccine Efficacy Trial
Barton F. Haynes,Peter B. Gilbert,M. Juliana McElrath,Susan Zolla-Pazner,Georgia D. Tomaras,S. Munir Alam,David T. Evans,David C. Montefiori,Chitraporn Karnasuta,Ruengpueng Sutthent,Hua-Xin Liao,Anthony L. DeVico,George K. Lewis,Constance Williams,Abraham Pinter,Youyi Fong,Holly Janes,Allan C. deCamp,Yunda Huang,Mangala Rao,Erik Billings,Nicos Karasavvas,Merlin L. Robb,Viseth Ngauy,Mark de Souza,Robert Paris,Guido Ferrari,Robert T. Bailer,Kelly A. Soderberg,Charla Andrews,Phillip W. Berman,Nicole Frahm,Stephen C. De Rosa,Michael D. Alpert,Nicole L. Yates,Xiaoying Shen,Richard A. Koup,Punnee Pitisuttithum,Jaranit Kaewkungwal,Sorachai Nitayaphan,Supachai Rerks-Ngarm,Nelson L. Michael,Jerome H. Kim +42 more
TL;DR: V vaccines that are designed to induce higher levels of V1V2 antibodies and lower levels of Env-specific IgA antibodies than are induced by the RV144 vaccine may have improved efficacy against HIV-1 infection.
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Protection of Macaques against Pathogenic Simian/Human Immunodeficiency Virus 89.6PD by Passive Transfer of Neutralizing Antibodies
John R. Mascola,Mark G. Lewis,Gabriela Stiegler,Dawn Harris,Thomas C. VanCott,Deborah Hayes,Mark K. Louder,Charles R. Brown,Christine V. Sapan,Sarah Schlesinger Frankel,Yichen Lu,Merlin L. Robb,Hermann Katinger,Deborah L. Birx +13 more
TL;DR: A general correlation between in vitro neutralization and protection is indicated and it is suggested that a vaccine that elicits neutralizing antibody should have a protective effect against HIV-1 infection or disease.
Journal ArticleDOI
Human skin Langerhans cells are targets of dengue virus infection.
Shuenn-Jue L. Wu,Geraldine Grouard-Vogel,Wellington Sun,John R. Mascola,Elena F. Brachtel,Elena F. Brachtel,Ravithat Putvatana,Mark K. Louder,Luis Filgueira,Mary A. Marovich,Henry K. Wong,Andrew Blauvelt,Gerald S. Murphy,Gerald S. Murphy,Merlin L. Robb,Bruce L. Innes,Deborah L. Birx,Curtis G. Hayes,Sarah Schlesinger Frankel,Sarah Schlesinger Frankel +19 more
TL;DR: Data demonstrate that human skin dendritic cells are permissive for DV infection, and provide a potential mechanism for the transmission of DV into human skin.