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Peter Arner

Researcher at Karolinska Institutet

Publications -  565
Citations -  56932

Peter Arner is an academic researcher from Karolinska Institutet. The author has contributed to research in topics: Adipose tissue & Lipolysis. The author has an hindex of 114, co-authored 553 publications receiving 52710 citations. Previous affiliations of Peter Arner include Karolinska University Hospital & Bristol-Myers Squibb.

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Journal ArticleDOI

In vivo observations on the lipolytic effect of noradrenaline during therapeutic fasting.

TL;DR: Fasting enhances the lipolytic effect of NA in vivo, and this effect is due to increased sensitivity to NA and also to NA-induced depression of the circulating insulin level.
Book ChapterDOI

Fatty acid binding protein expression in different human adipose tissue depots in relation to rates of lipolysis and insulin concentration in obese individuals

TL;DR: The relative ALBP/KLBP content of human adipose tissue is different in different adipose tissues depots and at the RNA level is related to the circulating insulin concentration, at least in obese subjects.
Journal ArticleDOI

Phosphodiesterase activity in human subcutaneous adipose tissue in insulin- and noninsulin-dependent diabetes mellitus.

TL;DR: The results suggest that the low Km PDE is inhibited in untreated IDDM and NIDDM, and PDE may be one factor responsible for regulation of the cAMP level.
Journal ArticleDOI

Effect of insulin on human adipose tissue metabolism in situ. Interactions with beta-adrenoceptors.

TL;DR: It is concluded that insulin action on glucose uptake and lipolysis in human adipose tissue in vivo is counteracted by beta-adrenoceptor stimulation, and insulin and beta- adrenoceptors have synergistic effects on non-oxidative glucose metabolism in human fat tissue in situ.
Journal ArticleDOI

Improved glucose metabolism after gastric bypass: evolution of the paradigm

TL;DR: Improved whole-body insulin sensitivity and postprandial glucose response occur early after Roux-en-Y gastric bypass, suggesting that the combination of different mechanisms is what makes RYGB an effective intervention for type 2 diabetes.