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Thomas F. Lüscher

Researcher at University of Zurich

Publications -  1613
Citations -  88517

Thomas F. Lüscher is an academic researcher from University of Zurich. The author has contributed to research in topics: Endothelium & Myocardial infarction. The author has an hindex of 134, co-authored 1560 publications receiving 79034 citations. Previous affiliations of Thomas F. Lüscher include University of Texas Southwestern Medical Center & Durham University.

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Journal Article

Calcium antagonists and ACE inhibitors : effect on endothelium and vascular smooth muscle. Discussion

TL;DR: Calcium antagonists and angiotensin conveting enzyme (ACE) inhibitors are widely used in patients with cardiovascular disease and are thought to have vascular protective effects.
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Misconceptions and Facts About Takotsubo Syndrome.

TL;DR: Takotsubo syndrome, initially described in the 1990s by Sato, has been increasingly recognized around the world and pathophysiology is directed towards central role of catecholamine surge, but other aspects like microvascular endothelial dysfunction and vasospasm have also been described.
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Stem cells in cardiovascular regeneration: from preservation of endogenous repair to future cardiovascular therapies.

TL;DR: This review summarizes current pre-clinical and clinical evidence for the potential role and mechanisms of action of stem and progenitor cells in vascular and cardiac repair and regeneration as well as recent developments in tissue engineering.
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High dose lisinopril in heart failure: economic considerations.

TL;DR: Based on the results of the ATLAS study, high doses of lisinopril are associated with a reduction in the number of hospitalizations for HF and for any reason and with a statistically non-significant reduction in mortality.
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Drug-eluting stents vs. bare metal stents in patients with cardiogenic shock: a comparison by propensity score analysis.

TL;DR: DESs appear to be associated with improved clinical outcomes, including a reduction in all-cause mortality compared with BMSs among patients undergoing PCI for cardiogenic shock, possibly because of a pacification of the infarct-related artery by anti-inflammatory drug.