T
Thomas F. Lüscher
Researcher at University of Zurich
Publications - 1613
Citations - 88517
Thomas F. Lüscher is an academic researcher from University of Zurich. The author has contributed to research in topics: Endothelium & Myocardial infarction. The author has an hindex of 134, co-authored 1560 publications receiving 79034 citations. Previous affiliations of Thomas F. Lüscher include University of Texas Southwestern Medical Center & Durham University.
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Journal ArticleDOI
Myeloid-related protein 8/14 complex is released by monocytes and granulocytes at the site of coronary occlusion: a novel, early, and sensitive marker of acute coronary syndromes.
Lukas Altwegg,Michel Neidhart,Martin Hersberger,Simone Müller,Franz R. Eberli,Roberto Corti,Marco Roffi,Gabor Sütsch,Arnold von Eckardstein,Manfred Wischnewsky,Thomas F. Lüscher,Willibald Maier +11 more
TL;DR: The occurrence of elevated MRP8/14 in the systemic circulation prior to markers of myocardial necrosis makes it a prime candidate for the detection of unstable plaques and management of ACS.
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2017 Update of ESC/EAS Task Force on practical clinical guidance for proprotein convertase subtilisin/kexin type 9 inhibition in patients with atherosclerotic cardiovascular disease or in familial hypercholesterolaemia
Ulf Landmesser,M. John Chapman,Jane K Stock,Pierre Amarenco,Jill J. F. Belch,Jan Borén,Michel Farnier,Brian A. Ference,Stephan Gielen,Ian D. Graham,Diederick E. Grobbee,G. Kees Hovingh,Thomas F. Lüscher,Massimo F Piepoli,Kausik K. Ray,Erik S.G. Stroes,Olov Wiklund,Stephan Windecker,José Luis Zamorano,Fausto J. Pinto,Lale Tokgozoglu,Jeroen J. Bax,Alberico L. Catapano +22 more
TL;DR: A correction has been published: European Heart Journal, Volume 39, Issue 22, 7 June 2018, pages 2105 and 2105.
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Endothelin-1 plasma levels in normal-tension glaucoma: abnormal response to postural changes
TL;DR: The hypothesis that vascular dysfunction may be involved in the pathogenesis of optic nerve damage in normal-tension glaucoma in NTG patients is supported.
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Long COVID: post-acute sequelae of COVID-19 with a cardiovascular focus
TL;DR: A possible model for referral of post-COVID-19 patients to cardiac services is proposed and future directions including research priorities and clinical trials that are currently underway to evaluate the efficacy of treatment strategies for long COVID and associated CV sequelae are discussed.
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Tetrahydrobiopterin and endothelial nitric oxide synthase activity
TL;DR: Data indicate that catabolism of NO by its reaction with superoxide could be an important mechanism underlying endothelial dysfunction and oxidative vascular injury described in a number of vascular diseases.