T
Thomas F. Lüscher
Researcher at University of Zurich
Publications - 1613
Citations - 88517
Thomas F. Lüscher is an academic researcher from University of Zurich. The author has contributed to research in topics: Endothelium & Myocardial infarction. The author has an hindex of 134, co-authored 1560 publications receiving 79034 citations. Previous affiliations of Thomas F. Lüscher include University of Texas Southwestern Medical Center & Durham University.
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Journal ArticleDOI
Spontaneous calcium-independent nitric oxide synthase activity in porcine ciliary processes.
TL;DR: The presence of a spontaneous calcium-independent NOS activity in the ciliary processes suggests that nitric oxide (NO) production is highly regulated in this tissue.
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Is there a rationale for combining angiotensinconverting enzyme inhibitors and calcium antagonists in cardiovascular disease
TL;DR: Long-term combination therapy of ACE inhibitors and calcium antagonists might provide beneficial effects in cardiovascular disease because they exert synergistic hemodynamic, antiproliferative, antithrombotic, and antiatherogenic properties.
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Human connective tissue growth factor expressed in Escherichia coli is a non-mitogenic inhibitor of apoptosis.
Daniel Gygi,Peter Zumstein,Doris Grossenbacher,Lukas Altwegg,Thomas F. Lüscher,Heinz Gehring +5 more
TL;DR: It is shown that in serum-deprived chicken embryo fibroblasts (CEF) the initial decline of cCTGF mRNA was followed by an increase, and CTGF supports very efficiently survival of certain cells without stimulating their cell growth.
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Soluble lectin-like oxidized low-density lipoprotein receptor-1 predicts premature death in acute coronary syndromes.
Simon Kraler,Florian A. Wenzl,Georgios Georgiopoulos,Slayman Obeid,Luca Liberale,Arnold von Eckardstein,Olivier Muller,François Mach,Lorenz Räber,Sylvain Losdat,Martin Schmiady,Konstantinos Stellos,Kimon Stamatelopoulos,Giovanni G. Camici,Annie Srdic,Francesco Lanni,Alexander Akhmedov,Thomas F. Lüscher +17 more
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Journal ArticleDOI
Heart non-specific effector CD4+ T cells protect from postinflammatory fibrosis and cardiac dysfunction in experimental autoimmune myocarditis.
Martina Zarak-Crnkovic,Gabriela Kania,Agnieszka Jaźwa-Kusior,Marcin Czepiel,Winandus J Wijnen,Jarosław Czyż,Björn Müller-Edenborn,Daria Vdovenko,Diana Lindner,Cristina Gil-Cruz,Marta Bachmann,Dirk Westermann,Burkhard Ludewig,Oliver Distler,Thomas F. Lüscher,Karin Klingel,Urs Eriksson,Przemyslaw Blyszczuk,Przemyslaw Blyszczuk +18 more
TL;DR: A proof-of-concept that heart non-specific Teff cells could effectively contribute to myocarditis and protect the heart from the dilated cardiomyopathy outcome is provided.