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Thomas F. Lüscher

Researcher at University of Zurich

Publications -  1613
Citations -  88517

Thomas F. Lüscher is an academic researcher from University of Zurich. The author has contributed to research in topics: Endothelium & Myocardial infarction. The author has an hindex of 134, co-authored 1560 publications receiving 79034 citations. Previous affiliations of Thomas F. Lüscher include University of Texas Southwestern Medical Center & Durham University.

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Endothelial Nitric Oxide Synthase Host Defense Enzyme of the Endothelium

TL;DR: The physiology of superoxide generation by endothelial nitric oxide synthase (eNOS) is explored, the so-called "uncoupled" state of the enzyme, and it is proposed that this uncoupled state may contribute to defense against infections.
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Real-Time Left Ventricular Pressure-Volume Loops During Percutaneous Mitral Valve Repair With the MitraClip System

TL;DR: Percutaneous mitral valve repair improves hemodynamic profiles and induces reverseleft ventricular remodeling by reducing left ventricular preload while preserving contractility and in nonsurgical candidates with compromised left Ventricular function, MitraClip therapy could be considered an alternative to surgical mitral valves repair.
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Takotsubo cardiomyopathy has a unique cardiac biomarker profile: NT-proBNP/myoglobin and NT-proBNP/troponin T ratios for the differential diagnosis of acute coronary syndromes and stress induced cardiomyopathy

TL;DR: Takotsubo cardiomyopathy goes along with a singular cardiac biomarker profile, which might be useful to identify patients with TC among patients presenting with acute coronary syndromes (ACS).
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Endothelium-Dependent Relaxations in Human Arteries

TL;DR: In contracted arterial rings, acetylcholine caused endothelium-dependent relaxations that were not inhibited by indomethacin in either artery but were significantly augmented in the renal artery, indicating that the relaxations are mediated by the release of an endotHelium-derived relaxing factor (or factors) rather than the releaseof prostacyclin.