T
Thomas F. Lüscher
Researcher at University of Zurich
Publications - 1613
Citations - 88517
Thomas F. Lüscher is an academic researcher from University of Zurich. The author has contributed to research in topics: Endothelium & Myocardial infarction. The author has an hindex of 134, co-authored 1560 publications receiving 79034 citations. Previous affiliations of Thomas F. Lüscher include University of Texas Southwestern Medical Center & Durham University.
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Journal ArticleDOI
Nitric oxide inhibits angiotensin II-induced migration of rat aortic smooth muscle cell. Role of cyclic-nucleotides and angiotensin1 receptors.
TL;DR: Findings indicate that NO inhibits AII-induced migration of vascular SMCs; the antimigratory effect of NO is mediated in part via a cGMP-dependent mechanism; and (c) AII stimulates SMC migration via an AT1 receptor.
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Expert consensus document from the European Society of Cardiology on catheter-based renal denervation
Felix Mahfoud,Thomas F. Lüscher,Bert Andersson,Iris Baumgartner,Renata Cifkova,Carlo DiMario,Pieter A. Doevendans,Robert Fagard,Jean Fajadet,Michel Komajda,Thierry Lefèvre,Chaim Lotan,Horst Sievert,Massimo Volpe,Massimo Volpe,Petr Widimsky,William Wijns,Bryan Williams,Stephan Windecker,Adam Witkowski,Thomas Zeller,Michael Böhm +21 more
TL;DR: In this paper, a consensus document summarizes the view of an expert panel of the European Society of Cardiology and the European Association of Percutaneous Cardiovascular Interventions to provide guidance regarding catheter-based renal denervation.
Journal ArticleDOI
Atherosclerosis and the two faces of endothelial nitric oxide synthase
TL;DR: There are several reasons to believe that NO synthesis could indeed be impaired in hypercholesterolemia and atherosclerosis, and major risk factors for atherosclerotic vascular disease have been associated with impaired NO activity.
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Mental stress induces prolonged endothelial dysfunction via endothelin-A receptors
Lukas E. Spieker,David Hürlimann,Frank Ruschitzka,Roberto Corti,Frank Enseleit,Sidney Shaw,Daniel Hayoz,John E. Deanfield,Thomas F. Lüscher,Georg Noll +9 more
TL;DR: Mental stress induces prolonged endothelial dysfunction, which is prevented by selective endothelin-A receptor antagonism, which represents a novel and important link between mental stress and atherosclerotic vascular disease.
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Human Connective Tissue Growth Factor Is Expressed in Advanced Atherosclerotic Lesions
Barry S. Oemar,Werner A,Garnier Jm,Do Dd,Godoy N,Markus Nauck,Winfried März,Rupp J,Pech M,Thomas F. Lüscher +9 more
TL;DR: It is shown for the first time that both hCTGF mRNA and protein are expressed in human arteries in vivo and that hCT GF may represent a novel factor expressed at high levels specifically in advanced lesions and may play a role in the development and progression of atherosclerosis.