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William E. Kraus

Researcher at Duke University

Publications -  625
Citations -  40583

William E. Kraus is an academic researcher from Duke University. The author has contributed to research in topics: Heart failure & Medicine. The author has an hindex of 93, co-authored 565 publications receiving 33692 citations. Previous affiliations of William E. Kraus include University of Texas Southwestern Medical Center & University of North Carolina at Chapel Hill.

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Lack of Association of a Functional Polymorphism in the Serotonin Receptor Gene With Body Mass Index and Depressive Symptoms in a Large Meta-Analysis of Population Based Studies

TL;DR: In the largest study of this polymorphism, it is determined that rs6318 is not associated with depression, or BMI, and a random effects meta-analysis was conducted to examine whether the Cys23Ser variant was associated with depressive symptoms and body mass index (BMI).
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Associations between neighborhood socioeconomic cluster and hypertension, diabetes, myocardial infarction, and coronary artery disease within a cohort of cardiac catheterization patients.

TL;DR: In this article, the authors used Ward's Hierarchical clustering to define eight neighborhood clusters across North Carolina (NC) using 11 census-based indicators of SES, race, housing, and urbanicity and assigned 6992 cardiac catheterization patients at Duke University Hospital from 2001-2010 to clusters.
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Field-Based Assessments of Behavioral Patterns During Shiftwork in Police Academy Trainees Using Wearable Technology

TL;DR: Findings show wearable technology combined with specialized data pre-processing can be used to monitor changes in behavioral patterns during shiftwork, and imputed labels of circadian misaligned sleep episodes that occurred during daytime, which were previously were mislabeled as non-sleep by Garmin.
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Circulating long chain acylcarnitines and outcomes in diabetic heart failure: an HF-ACTION clinical trial substudy.

TL;DR: In this paper, the authors used metabolomic profiling to identify biomarkers that report on exercise capacity, clinical outcomes, and differential response to exercise in heart failure patients with and without diabetes mellitus (DM).
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Rheumatoid arthritis complicates noninvasive whole blood gene expression testing for coronary artery disease.

TL;DR: Across a range of coronary artery disease severity, RA was associated with altered expression of CAD-associated genes, which highlight common pathogenic mechanisms for RA and CAD and validate the prior exclusion of RA patients from ASGES-based evaluation of CAD likelihood.