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William E. Kraus
Researcher at Duke University
Publications - 625
Citations - 40583
William E. Kraus is an academic researcher from Duke University. The author has contributed to research in topics: Heart failure & Medicine. The author has an hindex of 93, co-authored 565 publications receiving 33692 citations. Previous affiliations of William E. Kraus include University of Texas Southwestern Medical Center & University of North Carolina at Chapel Hill.
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Journal ArticleDOI
Association between satellite-based estimates of long-term PM2.5 exposure and coronary artery disease
Laura McGuinn,Cavin K. Ward-Caviness,Lucas M. Neas,Alexandra Schneider,David Diaz-Sanchez,Wayne E. Cascio,William E. Kraus,Elizabeth R. Hauser,Elaine Dowdy,Carol Haynes,Alexandra Chudnovsky,Petros Koutrakis,Robert B. Devlin +12 more
TL;DR: Satellite-based estimates of long-term PM2.5 exposure were associated with both coronary artery disease (CAD) and incidence of myocardial infarction (MI) in a cohort of cardiac catheterization patients.
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Effect of calorie restriction on the free-living physical activity levels of nonobese humans: results of three randomized trials
Corby K. Martin,Sai Krupa Das,Lauren Lindblad,Susan B. Racette,Megan A. McCrory,Edward P. Weiss,Edward P. Weiss,James P. DeLany,William E. Kraus +8 more
TL;DR: CR-associated changes in AEE were variable but, generally, reduced the energy deficit, which would reduce the expected rate of weight loss.
Journal ArticleDOI
Increased levels of apoptosis in gastrocnemius skeletal muscle in patients with peripheral arterial disease.
Robert G. Mitchell,Brian D. Duscha,Jennifer L. Robbins,Shelley I. Redfern,Jayer Chung,Daniel Bensimhon,William E. Kraus,William R. Hiatt,Judith G. Regensteiner,Brian H. Annex +9 more
TL;DR: PAD is associated with increased levels of apoptosis in the peripheral skeletal muscle, and further study is required to ascertain whether apoptosis plays a role in decreased functional capacity.
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Fine particulate matter and cardiovascular disease: Comparison of assessment methods for long-term exposure
Laura McGuinn,Cavin K. Ward-Caviness,Lucas M. Neas,Alexandra Schneider,Qian Di,Alexandra Chudnovsky,Joel Schwartz,Petros Koutrakis,Armistead G. Russell,Valerie Garcia,William E. Kraus,Elizabeth R. Hauser,Wayne E. Cascio,David Diaz-Sanchez,Robert B. Devlin +14 more
TL;DR: Long‐term air pollution exposure was associated with coronary artery disease for both modeled and monitored data and the elevated odds for CAD>23 and MI were nearly equivalent for all exposure assessment methods.
Journal ArticleDOI
Ozone exposure is associated with acute changes in inflammation, fibrinolysis, and endothelial cell function in coronary artery disease patients.
Jaime E. Mirowsky,Jaime E. Mirowsky,Martha Sue Carraway,Radhika Dhingra,Haiyan Tong,Lucas M. Neas,David Diaz-Sanchez,Wayne E. Cascio,Martin Case,James L. Crooks,Elizabeth R. Hauser,Elizabeth R. Hauser,Z. Elaine Dowdy,William E. Kraus,Robert B. Devlin +14 more
TL;DR: The results support the biological plausibility of ozone-induced cardiovascular effects and were found at concentrations below the EPA National Ambient Air Quality Standards for both ozone and PM2.5.