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Alejandra Bravo

Researcher at National Autonomous University of Mexico

Publications -  239
Citations -  14447

Alejandra Bravo is an academic researcher from National Autonomous University of Mexico. The author has contributed to research in topics: Bacillus thuringiensis & Cry1Ac. The author has an hindex of 58, co-authored 226 publications receiving 12942 citations. Previous affiliations of Alejandra Bravo include Pasteur Institute & Centre national de la recherche scientifique.

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Mode of action of Bacillus thuringiensis Cry and Cyt toxins and their potential for insect control.

TL;DR: Recent evidence suggests that Cyt synergize or overcome resistance to mosquitocidal-Cry proteins by functioning as a Cry-membrane bound receptor, and compares them to the mode of action of other bacterial PFT.
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Bacillus thuringiensis: A story of a successful bioinsecticide

TL;DR: Recently, similar Cry-binding proteins have been identified in the three insect orders, as cadherin, aminopeptidase-N and alkaline phosphatase suggesting a conserved mode of action, suggesting a significant reduction in chemical insecticide use.
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RNA interference in Lepidoptera: An overview of successful and unsuccessful studies and implications for experimental design

Olle Terenius, +78 more
TL;DR: Despite a large variation in the data, trends that are found are that RNAi is particularly successful in the family Saturniidae and in genes involved in immunity and that gene expression in epidermal tissues seems to be most difficult to silence.
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How Bacillus thuringiensis has evolved specific toxins to colonize the insect world

TL;DR: Assessment of phylogenetic relationships of the three domains of the active toxin and experimental results indicate how sequence divergence in combination with domain swapping by homologous recombination might have caused this extensive range of specificities.
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Bacillus thuringiensis insecticidal three‐domain Cry toxins: mode of action, insect resistance and consequences for crop protection

TL;DR: Activity of Cry1AMod toxins, which are able to form toxin oligomers in the absence of receptors, against different resistant populations, supports the hypothesis that toxin oligomerization is a limiting step in the Cry insecticidal activity.