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Arnold J. Levine
Researcher at Institute for Advanced Study
Publications - 493
Citations - 122094
Arnold J. Levine is an academic researcher from Institute for Advanced Study. The author has contributed to research in topics: Gene & Mutant. The author has an hindex of 139, co-authored 485 publications receiving 116005 citations. Previous affiliations of Arnold J. Levine include Harvard University & Affymetrix.
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Journal ArticleDOI
Plasma cell malignancy in the acquired immune deficiency syndrome. Association with Epstein-Barr virus.
Karl V. Voelkerding,Linda M. Sandhaus,Hugh C. Kim,Joanna B. Wilson,Tom Chittenden,Arnold J. Levine,Karel Raska +6 more
TL;DR: DNA analysis of autopsy-derived tumor tissues demonstrated clonal rearrangement of the immunoglobulin (Ig) heavy chain gene locus and rearrangements in both kappa and lambda light chain gene Loci.
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Deoxyribonucleic acid replication in simian virus 40-infected cells. II. Detection and characterization of simian virus 40 pseudovirions.
TL;DR: Pseudovirions are enriched, relative to true virions, on the lighter density side of infectious SV40 virus banded to equilibrium in a CsCl gradient.
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DNA replication of SV40-infected cells: VII. Formation of SV40 catenated and circular dimers
Rudolf Jaenisch,Arnold J. Levine +1 more
TL;DR: These data, together with previous results Jaeniseh & Levine 1972, are consistent with the hypothesis that catenated dimers arise by replication errors which are enhanced in the presence of cycloheximide.
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P53 and the defenses against genome instability caused by transposons and repetitive elements.
TL;DR: Genetic evidence for a piRNA‐mediated p53 interaction with transposons in Drosophila and zebrafish is presented, but it is herein placed in the context of a decade or so of additional work that demonstrated a role for p53 in regulatingTransposons and other repetitive elements.
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Cytotoxic T-Lymphocyte Epitope Immunodominance in the Control of Choroid Plexus Tumors in Simian Virus 40 Large T Antigen Transgenic Mice
Todd D. Schell,Lawrence M. Mylin,Ingo Georgoff,Angelica K. Teresky,Arnold J. Levine,Satvir S. Tevethia +5 more
TL;DR: It is demonstrated that SV11+ mice are functionally tolerant to the immunodominant Tag CTL epitopes, indicating that CTLs specific for the H-2Kb-restricted Tag epitope IV control the progressive growth of spontaneous tumors induced by this DNA virus oncogene in transgenic mice.