E
Edson X. Albuquerque
Researcher at University of Maryland, Baltimore
Publications - 268
Citations - 18565
Edson X. Albuquerque is an academic researcher from University of Maryland, Baltimore. The author has contributed to research in topics: Nicotinic agonist & Acetylcholine. The author has an hindex of 69, co-authored 268 publications receiving 17395 citations. Previous affiliations of Edson X. Albuquerque include Federal University of Rio de Janeiro & National Institute on Drug Abuse.
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Journal ArticleDOI
Freeze‐fracture, labelled‐replica, and electrophysiological studies of junctional fold destruction in my asthenia gravis and experimental autoimmune mysthenia gravis*
John E. Rash,C. Sue Hudson,Edson X. Albuquerque,Mohyee E. Eldefrawi,Richard F. Mayer,William Graham,Timothy J. A. Johnson,F. Dennis Giddings +7 more
TL;DR: The experiments described in this report were performed from 1975 to 1979 while all of the authors except Dr. Mayer and Mr. Giddings were members of the Department of Pharmacology and Experimental Therapeutics, University of Maryland School of Medicine.
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Pretreatment of Guinea Pigs with Galantamine Prevents Immediate and Delayed Effects of Soman on Inhibitory Synaptic Transmission in the Hippocampus
TL;DR: Testing the hypothesis that galantamine can prevent immediate and delayed effects of soman on hippocampal inhibitory synaptic transmission found it to be an important determinant of the antidotal effectiveness of galantamines.
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Strain-specific nicotinic modulation of glutamatergic transmission in the CA1 field of the rat hippocampus: August Copenhagen Irish versus Sprague-Dawley.
Manickavasagom Alkondon,Edna F. R. Pereira,Michelle C. Potter,Frederick C. Kauffman,Robert Schwarcz,Edson X. Albuquerque +5 more
TL;DR: The results indicate that the alpha3beta4* nAChR activity in glutamatergic neurons/axons and the number of glutamatorgic terminals synapsing onto CA1 SR interneurons are larger in prepubertal ACI than SD rats.
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Consequences of axonal transport blockade by batrachotoxin on mammalian neuromuscular junction. III. An ultrastructural study.
TL;DR: Normal physiological function and morphology were restored after fast axonal transport recovered and sufficient quantities of appropriate materials necessary for the maintenance of the nerve terminals and muscle membrane potential were again transported distally from the nerve cell body.
Journal Article
Interactions of phencyclidine with crayfish muscle membranes. Sensitivity to calcium channel antagonists and other drugs.
Esam E. El-Fakahany,Amira T. Eldefrawi,Deirdre Murphy,L G Aguayo,David J. Triggle,Edson X. Albuquerque,Mohyee E. Eldefrawi +6 more
TL;DR: Although verapamil and nifedipine inhibited theaction potential of crayfish muscle, PCP did not and actually prolonged slightly the falling phase of the action potential, suggesting that it is possible that the [3H]PCP binding protein in cray fish muscles is a Ca2+ channel.