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Enrique Amaya

Researcher at University of Manchester

Publications -  94
Citations -  7354

Enrique Amaya is an academic researcher from University of Manchester. The author has contributed to research in topics: Xenopus & Regeneration (biology). The author has an hindex of 37, co-authored 92 publications receiving 6955 citations. Previous affiliations of Enrique Amaya include University of North Carolina at Chapel Hill & Wellcome Trust/Cancer Research UK Gurdon Institute.

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Expression of a dominant negative mutant of the FGF receptor disrupts mesoderm formation in Xenopus embryos.

TL;DR: The results demonstrate that the FGF signaling pathway plays an important role in early embryogenesis, particularly in the formation of the posterior and lateral mesoderm.
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Transgenic Xenopus embryos from sperm nuclear transplantations reveal FGF signaling requirements during gastrulation

TL;DR: A simple approach for large-scale transgenesis in Xenopus laevis embryos is developed and used to identify in vivo requirements for FGF signaling during gastrulation and, by contrast, embryos expressing XFD contain well-patterned nervous systems despite a putative role for F GF in neural induction.
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The genome of the western clawed frog xenopus tropicalis

Uffe Hellsten, +49 more
- 30 Apr 2010 - 
TL;DR: The western clawed frog Xenopus tropicalis is an important model for vertebrate development that combines experimental advantages of the African clawed frogs Xenopus laevis with more tractable genetics.
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Amputation-induced reactive oxygen species are required for successful Xenopus tadpole tail regeneration

TL;DR: It is shown that Xenopus tadpole tail amputation induces a sustained production of reactive oxygen species (ROS) during tail regeneration, demonstrating that injury-induced ROS production is an important regulator of tissue regeneration.
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FGF signalling in the early specification of mesoderm in Xenopus.

TL;DR: Examination of the role of FGF signalling in the development of muscle and notochord and in the expression of early mesodermal markers in Xenopus embryos suggests that morphogenesis and tissue differentiation each depend on FGF and that the early dorsal mesoderm is already composed of two cell populations that differ in their requirements for FGF.