G
Georg N. Duda
Researcher at Charité
Publications - 613
Citations - 31004
Georg N. Duda is an academic researcher from Charité. The author has contributed to research in topics: Bone healing & Bone regeneration. The author has an hindex of 81, co-authored 563 publications receiving 25802 citations. Previous affiliations of Georg N. Duda include Humboldt University of Berlin & University of Ulm.
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Mechanosensation across borders: fibroblasts inside a macroporous scaffold sense and respond to the mechanical environment beyond the scaffold walls.
TL;DR: Surprisingly, the stiffness of the environment influenced cell behaviour even inside macroporous scaffolds where direct cell–cell contacts are hindered, and mechanical communication between cells via traction forces is essential for tissue adaptation to the environment and should not be blocked by rigid biomaterials.
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In Silico Biomechanical Evaluation of WE43 Magnesium Plates for Mandibular Fracture Fixation
TL;DR: In this paper , a 3D finite element model of the human mandible was developed, and four different fracture scenarios were simulated, together with physiological post-operative loading and boundary conditions.
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Treatment with recombinant human bone morphogenetic protein 7 leads to a transient induction of neutralizing autoantibodies in a subset of patients
Andrea Schuette,Arash Moghaddam,Petra Seemann,Georg N. Duda,Gerhard Schmidmaier,Lutz Schomburg +5 more
TL;DR: It is concluded that BMP7-aAB can be detected as natural aAB in healthy subjects, and are transiently induced by rhBMP7 therapy in a subset of patients.
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Accumulation of amino-polyvinyl alcohol-coated superparamagnetic iron oxide nanoparticles in bone marrow: implications for local stromal cells.
Frank Schulze,Azza Gramoun,Azza Gramoun,Lindsey A. Crowe,Anke Dienelt,Tugba Akcan,Heinrich Hofmann,Jean-Paul Vallée,Georg N. Duda,Andrea Ode +9 more
TL;DR: A-PVA-SPIONs appear suitable for contrast enhancement in bone marrow while the data suggest an influence on the BMSCs biology that necessitates future research.
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Bone morphogenetic protein 2-induced cellular chemotaxis drives tissue patterning during critical-sized bone defect healing: an in silico study
TL;DR: In this paper, the mechano-biological regulation of BMP-2-treated critical-sized bone defect healing was investigated using a multiphysics multiscale in silico approach.