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Hasan Obeid

Researcher at Paris Descartes University

Publications -  27
Citations -  93

Hasan Obeid is an academic researcher from Paris Descartes University. The author has contributed to research in topics: Internal medicine & Arterial stiffness. The author has an hindex of 3, co-authored 12 publications receiving 56 citations. Previous affiliations of Hasan Obeid include Ghent University & French Institute of Health and Medical Research.

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Towards a consensus on the understanding and analysis of the pulse waveform: Results from the 2016 Workshop on Arterial Hemodynamics: Past, present and future

TL;DR: Overall the responses and comments show a high measure of quantitative agreement with the various proposed 'consensus' statements and seem a useful basis for proceeding with a more detailed and comprehensive consensus document on the current understanding and approaches to analysis of the pulse waveform.
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Numerical assessment and comparison of pulse wave velocity methods aiming at measuring aortic stiffness

TL;DR: Computer model-based analyses demonstrate that for PWV methods based on peripheral signals, pulse transit time differences closely correlate with the aortic transit time, supporting the use of these methods in clinical practice.
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Arterial Stiffness: A New Biomarker to be Measured

TL;DR: Several measurement techniques are presented and an explanation of the link between cardiovascular events and arterial stiffness are summarized.
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Radial-digital pulse wave velocity: a noninvasive method for assessing stiffness of small conduit arteries

TL;DR: The interaction between the stiffness of various arterial segments is important in understanding the behavior of pressure and flow waves along the arterial tree in this paper, where the authors provide a novel method to measure the stiffness.
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Assessment of Stiffness of Large to Small Arteries in Multistage Renal Disease Model: A Numerical Study

TL;DR: The findings suggest that the PWV variations observed in clinical practice with different stages of kidney disease may be partially attributed to biomechanical alterations of the arterial tree and their effect on BP.