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Jonathan Z. Long

Researcher at Stanford University

Publications -  83
Citations -  12581

Jonathan Z. Long is an academic researcher from Stanford University. The author has contributed to research in topics: JZL184 & Fatty acid amide hydrolase. The author has an hindex of 37, co-authored 69 publications receiving 10665 citations. Previous affiliations of Jonathan Z. Long include Kettering University & Harvard University.

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Selective blockade of 2-arachidonoylglycerol hydrolysis produces cannabinoid behavioral effects

TL;DR: 2-AG endogenously modulates several behavioral processes classically associated with the pharmacology of cannabinoids and point to overlapping and unique functions for 2-AG and anandamide in vivo, indicating a functional segregation of endocannabinoid signaling pathways in vivo.
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Monoacylglycerol Lipase Regulates a Fatty Acid Network that Promotes Cancer Pathogenesis

TL;DR: Overexpression of MAGL in nonaggressive cancer cells recapitulates this fatty acid network and increases their pathogenicity-phenotypes that are reversed by an MAGL inhibitor, indicating that exogenous sources of fatty acids can contribute to malignancy in cancers lacking MAGL activity.
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Meteorin-like Is a Hormone that Regulates Immune-Adipose Interactions to Increase Beige Fat Thermogenesis

TL;DR: The identification of meteorin-like (Metrnl), a circulating factor that is induced in muscle after exercise and in adipose tissue upon cold exposure, which links host-adaptive responses to the regulation of energy homeostasis and tissue inflammation and has therapeutic potential for metabolic and inflammatory diseases.
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Endocannabinoid Hydrolysis Generates Brain Prostaglandins That Promote Neuroinflammation

TL;DR: These findings identify MAGL as a distinct metabolic node that couples endocannabinoid to prostaglandin signaling networks in the nervous system and suggest that inhibition of this enzyme may be a new and potentially safer way to suppress the proinflammatory cascades that underlie neurodegenerative disorders.