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Mark J. Lowe

Researcher at Cleveland Clinic

Publications -  158
Citations -  13249

Mark J. Lowe is an academic researcher from Cleveland Clinic. The author has contributed to research in topics: Resting state fMRI & Functional magnetic resonance imaging. The author has an hindex of 51, co-authored 150 publications receiving 12081 citations. Previous affiliations of Mark J. Lowe include University of Wisconsin-Madison & Indiana University – Purdue University Indianapolis.

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Toward discovery science of human brain function

Bharat B. Biswal, +54 more
TL;DR: The 1000 Functional Connectomes Project (Fcon_1000) as discussed by the authors is a large-scale collection of functional connectome data from 1,414 volunteers collected independently at 35 international centers.
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Functional Connectivity in Single and Multislice Echoplanar Imaging Using Resting-State Fluctuations

TL;DR: Correlations to left hemisphere motor cortex, visual cortex, and amygdala are measured in long resting-state scans and these correlations are extended to lower sampling rate multislice echoplanar acquisitions and other right/left hemisphere-symmetric functional cortices.
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Activity and Connectivity of Brain Mood Regulating Circuit in Depression: A Functional Magnetic Resonance Study

TL;DR: The finding of increased activation of limbic regions and decreased LFBF correlations between ACC and limbic areas is consistent with the hypothesis that decreased cortical regulation of limbics activation in response to negative stimuli may be present in depression.
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Resting state corticolimbic connectivity abnormalities in unmedicated bipolar disorder and unipolar depression

TL;DR: The results of the study indicate a common finding of decreased corticolimbic functional connectivity in different types of mood disorders.
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Antidepressant Effect on Connectivity of the Mood-Regulating Circuit: An fMRI Study

TL;DR: The results of this study are consistent with the hypothesis that antidepressant treatment may increase corticolimbic connectivity, thereby possibly increasing the regulatory influence of cortical mood-regulating regions over limbic regions.