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Martin R Turner

Researcher at University of Oxford

Publications -  519
Citations -  40655

Martin R Turner is an academic researcher from University of Oxford. The author has contributed to research in topics: Amyotrophic lateral sclerosis & RNA-binding protein. The author has an hindex of 98, co-authored 503 publications receiving 34965 citations. Previous affiliations of Martin R Turner include University College London & Drexel University.

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Requirement of bic/microRNA-155 for normal immune function.

TL;DR: It is shown that mice deficient for bic/microRNA-155 are immunodeficient and display increased lung airway remodeling, and suggests that bic-micro RNA-155 plays a key role in the homeostasis and function of the immune system.
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SeminarAmyotrophic lateral sclerosis

TL;DR: This paper summarized current concepts about the origin of the disease, what predisposes patients to develop the disorder, and discuss why all cases of ALS are not the same, focusing on the small steps of extending longevity, improving therapies, undertaking clinical trials, and compiling population registries to the overarching goals of establishing the measures that guard against onset and finding the triggers for this neurodegenerative disorder.
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Neurological and neuropsychiatric complications of COVID-19 in 153 patients: a UK-wide surveillance study.

Aravinthan Varatharaj, +125 more
TL;DR: This is the first nationwide, cross-specialty surveillance study of acute neurological and psychiatric complications of COVID-19 and provides valuable and timely data that are urgently needed by clinicians, researchers, and funders.
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Excessive production of interleukin 6/B cell stimulatory factor‐2 in rheumatoid arthritis

TL;DR: The data indicate that IL6 is generated constitutively in RA and its overproduction may explain the local as well as the generalized symptoms of RA, since IL6 can function as B cell growth and differentiation factor aswell as hepatocyte‐stimulating factor.
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microRNA-155 Regulates the Generation of Immunoglobulin Class-Switched Plasma Cells

TL;DR: The intrinsic requirement for miR-155 is shown in B cell responses to thymus-dependent and -independent antigens and implicate post-transcriptional regulation of gene expression for establishing the terminal differentiation program of B cells.