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Natasha L. Williams
Researcher at Australian Institute of Tropical Health and Medicine
Publications - 18
Citations - 857
Natasha L. Williams is an academic researcher from Australian Institute of Tropical Health and Medicine. The author has contributed to research in topics: Burkholderia pseudomallei & Melioidosis. The author has an hindex of 11, co-authored 17 publications receiving 777 citations. Previous affiliations of Natasha L. Williams include James Cook University.
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Journal ArticleDOI
Predictability of biomass burning in response to climate changes
Anne-Laure Daniau,Patrick J. Bartlein,Sandy P. Harrison,Sandy P. Harrison,Iain Colin Prentice,Iain Colin Prentice,Iain Colin Prentice,Scott Brewer,Pierre Friedlingstein,Pierre Friedlingstein,T I Harrison-Prentice,Jun Inoue,Kenji Izumi,Jennifer R. Marlon,Scott Mooney,Mitchell J. Power,Janelle Stevenson,Willy Tinner,Maja Andrič,Juliana Atanassova,Hermann Behling,M.P. Black,Olivier Blarquez,K. J. Brown,K. J. Brown,Christopher Carcaillet,Eric A. Colhoun,Daniele Colombaroli,Basil A. S. Davis,Donna D'Costa,John Dodson,Lydie M Dupont,Zewdu Eshetu,Daniel G. Gavin,Aurélie Genries,Simon Haberle,Douglas J. Hallett,Geoffrey Hope,Sally P. Horn,T.G. Kassa,Fumitaka Katamura,Lisa M. Kennedy,Peter Kershaw,Sergey K. Krivonogov,Colin J. Long,Donatella Magri,Elena Marinova,Elena Marinova,G.M. Mckenzie,Patricio I. Moreno,Patrick Moss,Frank H. Neumann,Frank H. Neumann,Elin Norström,C. Paitre,Damien Rius,Damien Rius,Neil Roberts,Guy Robinson,Naoko Sasaki,Louis Scott,Hikaru Takahara,Valery T. Terwilliger,Valery T. Terwilliger,Florian Thevenon,Rebecca Turner,Verushka Valsecchi,Verushka Valsecchi,Boris Vannière,Megan K. Walsh,Megan K. Walsh,Natasha L. Williams,Yunlin Zhang +72 more
TL;DR: This article analyzed sedimentary charcoal records to show that the changes in fire regime over the past 21,000 yrs are predictable from changes in regional climates and showed that fire increases monotonically with changes in temperature and peaks at intermediate moisture levels.
Journal ArticleDOI
Late Quaternary fire regimes of Australasia
Scott Mooney,Sandy P. Harrison,Patrick J. Bartlein,Anne-Laure Daniau,Janelle Stevenson,Katherine C. Brownlie,Solomon Buckman,Matthew L. Cupper,Jonathon Luly,M.P. Black,Eric A. Colhoun,Donna D'Costa,John Dodson,Simon Haberle,Geoffrey Hope,Peter Kershaw,Christine Kenyon,M. McKenzie,Natasha L. Williams +18 more
TL;DR: In this article, the authors examined the temporal and spatial variability of fire regimes during the Late Quaternary and found that fire in Australasia predominantly reflects climate, with colder periods characterized by less and warmer intervals by more biomass burning.
Journal ArticleDOI
Susceptibility to Burkholderia pseudomallei is associated with host immune responses involving tumor necrosis factor receptor-1 (TNFR1) and TNF receptor-2 (TNFR2).
TL;DR: It is demonstrated that in vivo depletion of macrophages renders C57BL/6 mice highly susceptible to intranasal infection with B. pseudomallei, with significant mortality occurring within 5 days of infection, and it is shown that TNF-alpha and both TNFR1 and TNFR2 are required for optimal control of the bacterium.
Journal ArticleDOI
Impact of streptozotocin-induced diabetes on functional responses of dendritic cells and macrophages towards Burkholderia pseudomallei.
TL;DR: It is demonstrated that diabetic mice with extended periods of uncontrolled hyperglycaemia have impaired DC and macrophage function towards B. pseudomallei, which may contribute to the high susceptibility observed in clinical practice.
Journal ArticleDOI
Group G Streptococcus Induces an Autoimmune Carditis Mediated by Interleukin 17A and Interferon γ in the Lewis Rat Model of Rheumatic Heart Disease.
Suchandan Sikder,Natasha L. Williams,Alanna E. Sorenson,Abdul Alim,Miranda E Vidgen,Nicole J. Moreland,Catherine M. Rush,Robert S Simpson,Brenda Govan,Robert Norton,Madeleine W. Cunningham,David J. McMillan,Kadaba S. Sriprakash,Natkunam Ketheesan +13 more
TL;DR: It is discovered that GGS causes interleukin 17A/interferon γ-induced myocarditis and valvulitis, hallmarks of ARF/RHD, and the histological, immunological, and functional changes in the hearts of rats exposed to GGS are identical to those exposure to GAS.