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Raymond J. Dolan

Researcher at University College London

Publications -  940
Citations -  150202

Raymond J. Dolan is an academic researcher from University College London. The author has contributed to research in topics: Prefrontal cortex & Functional magnetic resonance imaging. The author has an hindex of 196, co-authored 919 publications receiving 138540 citations. Previous affiliations of Raymond J. Dolan include VU University Amsterdam & McGovern Institute for Brain Research.

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Dopamine, affordance and active inference.

TL;DR: This paper focuses on the consequences of changing tonic levels of dopamine firing using simulations of cued sequential movements and uses these simulations to demonstrate how a single functional role for dopamine at the synaptic level can manifest in different ways at the behavioural level.
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Neural activity associated with episodic memory for emotional context.

TL;DR: Findings suggest that neural activity mediating episodic retrieval of contextual information and its subsequent processing is modulated by emotion in at least two ways: first, there is enhancement of activity in networks supporting episodic retrieved neutral information, and second, regions known to be activated when emotional information is encountered in the environment are also active when emotionalInformation is retrieved from memory.
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Human Pavlovian–Instrumental Transfer

TL;DR: This work provides a demonstration of behavioral pavlovian–instrumental transfer in humans, with an audiovisual predictor of the noncontingent delivery of money inducing participants to perform more avidly an action involving squeezing a handgrip to earn money.
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The role of the prefrontal cortex in higher cognitive functions

TL;DR: Brain imaging studies show that "holding something in mind" is associated with activity in an extended system which involves both prefrontal cortex and more posterior areas whose location is determined by the nature of the information being held in mind.
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Neural Origins of Human Sickness in Interoceptive Responses to Inflammation

TL;DR: These findings suggest that peripheral infection selectively influences central nervous system function to generate core symptoms of sickness and reorient basic motivational states.