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Raymond J. Dolan

Researcher at University College London

Publications -  940
Citations -  150202

Raymond J. Dolan is an academic researcher from University College London. The author has contributed to research in topics: Prefrontal cortex & Functional magnetic resonance imaging. The author has an hindex of 196, co-authored 919 publications receiving 138540 citations. Previous affiliations of Raymond J. Dolan include VU University Amsterdam & McGovern Institute for Brain Research.

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Differential encoding of losses and gains in the human striatum

TL;DR: It is shown that striatal activation reflects positively signed prediction errors for both financial reward and loss, and functional segregation within the striatum is shown, with more anterior regions showing relative selectivity for rewards and more posterior regions for losses.
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Anterior cingulate activity during error and autonomic response

TL;DR: Scanned subjects using functional magnetic resonance imaging while they performed numerical versions of the Stroop task suggested that an interface exists within ACC between cognitive and biobehavioral systems in the service of response adaptation, suggesting that ACC supports a generation of integrated bodily responses.
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Oxytocin Attenuates Affective Evaluations of Conditioned Faces and Amygdala Activity

TL;DR: The data suggest that oxytocin modulates the expression of evaluative conditioning for socially relevant faces via influences on amygdala and fusiform gyrus, an effect that may explain its prosocial effects.
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The Neural Basis of Mood-Congruent Processing Biases in Depression

TL;DR: These findings suggest a distinct neural substrate for mood-congruent processing biases in performance and suggest the medial and orbital prefrontal regions may play a key role in mediating the interaction between mood and cognition in affective disorder.
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Regional brain activity in chronic schizophrenic patients during the performance of a verbal fluency task.

TL;DR: Chronic schizophrenic patients can show a normal magnitude of frontal activation when matched for performance with controls, and they fail to show the expected reductions of activity in the superior temporal cortex, which may reflect abnormal functional connectivity between frontal and temporal cortex.