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Robin M. Murray

Researcher at King's College London

Publications -  1583
Citations -  128883

Robin M. Murray is an academic researcher from King's College London. The author has contributed to research in topics: Psychosis & Schizophrenia. The author has an hindex of 171, co-authored 1539 publications receiving 116362 citations. Previous affiliations of Robin M. Murray include University of Cambridge & National Institutes of Health.

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Meta-analysis of the P300 and P50 waveforms in schizophrenia

TL;DR: This meta-analysis confirms the existence of ERP deficits in schizophrenia, and the magnitude of these deficits is similar to the most robust findings reported in neuroimaging and neuropsychology in schizophrenia.
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Testing hypotheses on specific environmental causal effects on behavior

TL;DR: In this paper, the authors discuss the need to consider sample selection and the value of longitudinal data in order to test hypotheses on specific environmental risk mechanisms for psychopathology and conclude that environmental risk hypotheses can be put to the test but that it is usually necessary to use a combination of research strategies.
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Incidence of schizophrenia and other psychoses in ethnic minority groups: results from the MRC AESOP study

TL;DR: Findings suggest that either additional risk factors are operating in African-Caribbeans and Black Africans or that these factors are particularly prevalent in these groups, and that such factors increase risk for schizophrenia and mania inThese groups.
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Static and dynamic cognitive deficits in childhood preceding adult schizophrenia: a 30-year study.

TL;DR: The findings suggest that the origins of schizophrenia include two interrelated developmental processes evident from childhood to early adolescence (ages 7-13 years).
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Heterogeneity in incidence rates of schizophrenia and other psychotic syndromes: Findings from the 3-center ÆSOP study

TL;DR: There is significant and independent variation of incidence of schizophrenia and other psychoses in terms of sex, age, ethnicity, and place, which confirms that environmental effects at the individual, and perhaps neighborhood level, may interact together and with genetic factors in the etiology of psychosis.