R
Robin M. Murray
Researcher at King's College London
Publications - 1583
Citations - 128883
Robin M. Murray is an academic researcher from King's College London. The author has contributed to research in topics: Psychosis & Schizophrenia. The author has an hindex of 171, co-authored 1539 publications receiving 116362 citations. Previous affiliations of Robin M. Murray include University of Cambridge & National Institutes of Health.
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Journal ArticleDOI
Abnormal cortisol levels during the day and cortisol awakening response in first-episode psychosis: The role of stress and of antipsychotic treatment
Valeria Mondelli,Paola Dazzan,Nilay Hepgul,Marta Di Forti,Monica Aas,Alessandro D'Albenzio,Marco Di Nicola,Helen L. Fisher,Rowena Handley,Tiago Reis Marques,Craig Morgan,Serena Navari,Heather Taylor,Andrew Papadopoulos,Katherine J. Aitchison,Robin M. Murray,Carmine M. Pariante +16 more
TL;DR: It is shown that antipsychotics normalize diurnal cortisol hyper-secretion but not the blunted cortisol awakening response in FEP; factors other than the excess of psychosocial stress explain HPA axis abnormalities in F EP.
Journal ArticleDOI
An integrated genetic-epigenetic analysis of schizophrenia : evidence for co-localization of genetic associations and differential DNA methylation
Eilis Hannon,Emma Dempster,Joana Viana,Joe Burrage,Adam Smith,Ruby Macdonald,David St Clair,Colette J Mustard,Gerome Breen,Sebastian Therman,Jaakko Kaprio,Jaakko Kaprio,Timothea Toulopoulou,Hilleke E. Hulshoff Pol,Marc M. Bohlken,René S. Kahn,Igor Nenadic,Christina M. Hultman,Robin M. Murray,David A. Collier,David A. Collier,Nick Bass,Hugh Gurling,Andrew McQuillin,Leonard C. Schalkwyk,Leonard C. Schalkwyk,Jonathan Mill,Jonathan Mill,Jonathan Mill +28 more
TL;DR: This paper performed a multi-stage epigenome-wide association study, quantifying genome-wide patterns of DNA methylation in a total of 1714 individuals from three independent sample cohorts.
Journal ArticleDOI
Copy number variations of chromosome 16p13.1 region associated with schizophrenia
Andres Ingason,Andres Ingason,Dan Rujescu,Sven Cichon,Engilbert Sigurdsson,Thordur Sigmundsson,Olli Pietiläinen,Jacobine E. Buizer-Voskamp,Eric Strengman,Clyde Francks,Pierandrea Muglia,Arnaldur Gylfason,Omar Gustafsson,Pall I. Olason,Stacy Steinberg,Thomas Hansen,K.D. Jakobsen,Henrik B. Rasmussen,Ina Giegling,H.-J. Möller,Annette M. Hartmann,Caroline Crombie,Gillian Fraser,Neil Walker,Jan-Erik Lönnqvist,Jaana Suvisaari,Annamari Tuulio-Henriksson,Elvira Bramon,Lambertus A. Kiemeney,Barbara Franke,Robin M. Murray,Evangelos Vassos,Timothea Toulopoulou,Thomas W. Mühleisen,Sarah Tosato,Mirella Ruggeri,Srdjan Djurovic,Ole A. Andreassen,Zhongyang Zhang,Thomas Werge,Roel A. Ophoff,Roel A. Ophoff,M. Rietschel,Markus M. Nöthen,H. Petursson,Hreinn Stefansson,Leena Peltonen,Leena Peltonen,David A. Collier,Kari Stefansson,D. St Clair +50 more
TL;DR: It is concluded that duplications and perhaps also deletions of chromosome 16p13.1, previously reported to be associated with autism and MR, also confer risk of schizophrenia.
Journal ArticleDOI
Towards an aetiological classification of schizophrenia.
TL;DR: Although the aetiology of schizophrenia comprises genetic and environmental components acting in variable proportions, a simple division into familial and sporadic cases would facilitate research.
BookDOI
The epidemiology of schizophrenia
TL;DR: The implications of epidemiology for service planning in schizophrenia and gene-environment interaction in schizophrenia using neuroimaging are discussed, as well as the relationship between substance abuse and schizophrenia.