R
Robin M. Murray
Researcher at King's College London
Publications - 1583
Citations - 128883
Robin M. Murray is an academic researcher from King's College London. The author has contributed to research in topics: Psychosis & Schizophrenia. The author has an hindex of 171, co-authored 1539 publications receiving 116362 citations. Previous affiliations of Robin M. Murray include University of Cambridge & National Institutes of Health.
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Marijuana and Madness
TL;DR: I was left with the impression that marijuana is a very exciting and underused drug in neuroscience, as well as in other medical areas, and the authors have only scratched the surface of its many benefits.
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Neurocognitive performance in children aged 9-12 years who present putative antecedents of schizophrenia.
Alexis E. Cullen,Hannah Dickson,Sophie West,Robin G. Morris,Glenn L. Mould,Sheilagh Hodgins,Robin M. Murray,Kristin R. Laurens,Kristin R. Laurens +8 more
TL;DR: Children aged 9-12 years who present multiple antecedents of schizophrenia display poorer neurocognition than healthy peers on several domains showing pronounced deficits in schizophrenia, first-episode psychosis, and youth with prodromal symptoms.
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White matter microstructural abnormalities in families multiply affected with bipolar I disorder: a diffusion tensor tractography study
Louise Emsell,Chris Chaddock,Natalie J. Forde,W. Van Hecke,Gareth J. Barker,Alexander Leemans,Stefan Sunaert,Muriel Walshe,Elvira Bramon,Dara M. Cannon,Robin M. Murray,Colm McDonald +11 more
TL;DR: WM microstructural abnormalities in limbic, temporal and callosal pathways represent micro structural abnormalities associated with BD whereas alterations in the SLF and UF may represent potential markers of endophenotypic risk.
Journal Article
Schizophrenia and bipolar disorder: similarities in pathogenic mechanisms but differences in neurodevelopment.
TL;DR: Genes involved in early cortical development and early neurodevelopmental insults causing developmental impairment may put individuals on a trajectory towards schizophrenia rather than bipolar illness, which is the most plausible explanation.
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Structural covariance in the cortex of very preterm adolescents: a voxel-based morphometry study
Chiara Nosarti,Andrea Mechelli,Aimee Herrera,Muriel Walshe,Sukhwinder S. Shergill,Robin M. Murray,Larry Rifkin,Matthew Allin +7 more
TL;DR: Very preterm adolescents compared with controls demonstrated differential structural covariance between medial, frontal and cingulate gyri, caudate nucleus, thalamus, primary visual cortex, cerebellum and several other cortical and subcortical regions of the cortex, suggesting that developmental changes in one brain region may result in a cascade of alterations in multiple regions.