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Robin M. Murray

Researcher at King's College London

Publications -  1583
Citations -  128883

Robin M. Murray is an academic researcher from King's College London. The author has contributed to research in topics: Psychosis & Schizophrenia. The author has an hindex of 171, co-authored 1539 publications receiving 116362 citations. Previous affiliations of Robin M. Murray include University of Cambridge & National Institutes of Health.

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Randomized Controlled Trial of Effect of Prescription of Clozapine Versus Other Second-Generation Antipsychotic Drugs in Resistant Schizophrenia

TL;DR: In people with schizophrenia with poor treatment response to 2 or more antipsychotic drugs, there is an advantage to commencing clozapine rather than other SGA drugs in terms of symptom improvement over 1 year.
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Premorbid social functioning in schizophrenia and bipolar disorder: similarities and differences.

TL;DR: The data support the view that poor premorbid social adjustment is one manifestation of vulnerability to adult psychotic disorders and are consistent with other findings pointing to early developmental deficits in patients who subsequently develop psychosis.
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Pharmacogenetic prediction of clozapine response.

TL;DR: A combination of six polymorphisms in neurotransmitter-receptor-related genes resulted in 76.7% success in the prediction of clozapine response and a sensitivity of 95% (+/- 0.04) for satisfactory response in schizophrenic patients.
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Cognitive and motor function and the size of the cerebellum in adolescents born very pre-term.

TL;DR: There were significant associations between cerebellar volume and several cognitive test scores, in particular the Wechsler Intelligence Scale for Children-Revised, the Kaufman Assessment Battery for Children and the Schonnel reading age, which provides further evidence implicating the cerebellum in cognition.
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Abnormal monitoring of inner speech: a physiological basis for auditory hallucinations.

TL;DR: It is suggested that a predisposition to verbal hallucinations is associated with a failure to activate areas concerned with the monitoring of inner speech.