R
Robin M. Murray
Researcher at King's College London
Publications - 1583
Citations - 128883
Robin M. Murray is an academic researcher from King's College London. The author has contributed to research in topics: Psychosis & Schizophrenia. The author has an hindex of 171, co-authored 1539 publications receiving 116362 citations. Previous affiliations of Robin M. Murray include University of Cambridge & National Institutes of Health.
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Patterns of deficits in brain function in bipolar disorder and schizophrenia: a cluster analytic study.
Mei-Hua Hall,Jordan W. Smoller,Nancy R. Cook,Katja Schulze,Phil Lee,Grantley Taylor,Elvira Bramon,Michael J. Coleman,Robin M. Murray,Dean F. Salisbury,Deborah L. Levy +10 more
TL;DR: It is hypothesized that each neurophysiology subgroup may share similar genotypic profiles, which may increase statistical power to detect genetic risk factors in potentially biologically relevant homogenous subgroups independent of diagnostic boundaries.
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The dexamethasone suppression test in schizophrenia
TL;DR: R rates of dexamethasone non-suppression were very low; together with the high rates of depression, this suggests that depression in schizophrenia may have a different neuroendocrine profile from major depressive disorders.
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Interaction between DRD2 and AKT1 genetic variations on risk of psychosis in cannabis users: a case-control study.
Marco Colizzi,Conrad Iyegbe,John Powell,Giuseppe Blasi,Alessandro Bertolino,Robin M. Murray,Marta Di Forti +6 more
TL;DR: The genetic pathway × cannabis use interaction model was expected to better predict the individual’s odds of psychotic disorder than the single candidate gene×cannabis use interactionmodel.
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Continuity of care and clinical outcomes in the community for people with severe mental illness
TL;DR: Evidence of declining continuity of care in people with schizophrenia is found, and of an independent effect of this on worse clinical outcomes, which is suggested to be related to reorganisation of services.
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Early mortality in the megaloblastic anaemias.
TL;DR: It is suggested that in severe megaloblastic anaemia, polyuria and diarrhoea may lead to depletion of whole-body potassium and that with replacement therapy the sudden cellular demand causes a rapid fall in extracellular potassium.