S
Satoshi Fukuyama
Researcher at University of Tokyo
Publications - 68
Citations - 4339
Satoshi Fukuyama is an academic researcher from University of Tokyo. The author has contributed to research in topics: Virus & Influenza A virus. The author has an hindex of 33, co-authored 68 publications receiving 3839 citations. Previous affiliations of Satoshi Fukuyama include South Korean Ministry for Health, Welfare and Family Affairs & La Jolla Institute for Allergy and Immunology.
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Journal ArticleDOI
NALT- versus PEYER'S-patch-mediated mucosal immunity
Hiroshi Kiyono,Satoshi Fukuyama +1 more
TL;DR: A greater understanding of the differences between NALT and other organized lymphoid tissues, such as Peyer's patches, should facilitate the development of nasal vaccines.
Journal ArticleDOI
Characterization of H7N9 influenza A viruses isolated from humans
Tokiko Watanabe,Maki Kiso,Satoshi Fukuyama,Noriko Nakajima,Masaki Imai,Shinya Yamada,Shin Murakami,Seiya Yamayoshi,Kiyoko Iwatsuki-Horimoto,Yoshihiro Sakoda,Emi Takashita,Ryan McBride,Takeshi Noda,Masato Hatta,Hirotaka Imai,Dongming Zhao,Noriko Kishida,Masayuki Shirakura,Robert P. de Vries,Shintaro Shichinohe,Masatoshi Okamatsu,Tomokazu Tamura,Yuriko Tomita,Naomi Fujimoto,Kazue Goto,Hiroaki Katsura,Eiryo Kawakami,Izumi Ishikawa,Shinji Watanabe,Mutsumi Ito,Yuko Sakai-Tagawa,Yukihiko Sugita,Ryuta Uraki,Reina Yamaji,Amie J. Eisfeld,Gongxun Zhong,Shufang Fan,Jihui Ping,Eileen A. Maher,Anthony Hanson,Yuko Uchida,Takehiko Saito,Makoto Ozawa,Gabriele Neumann,Hiroshi Kida,Takato Odagiri,James C. Paulson,Hideki Hasegawa,Masato Tashiro,Yoshihiro Kawaoka +49 more
TL;DR: The robust replicative ability in mice, ferrets and nonhuman primates and the limited transmissibility in ferrets of Anhui/1 suggest that A(H7N9) viruses have pandemic potential.
Journal ArticleDOI
The pathogenesis of influenza virus infections: the contributions of virus and host factors
TL;DR: This work reviews the contributions of both virus and host factors to the pathogenesis of these viral infections and identifies several virulence factors in these virus strains.
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Influenza Virus-Host Interactome Screen as a Platform for Antiviral Drug Development
Tokiko Watanabe,Eiryo Kawakami,Jason E. Shoemaker,Tiago J. S. Lopes,Yukiko Matsuoka,Yuriko Tomita,Hiroko Kozuka-Hata,Takeo Gorai,Tomoko Kuwahara,Eiji Takeda,Atsushi Nagata,Ryo Takano,Maki Kiso,Makoto Yamashita,Yuko Sakai-Tagawa,Hiroaki Katsura,Naoki Nonaka,Hiroko Fujii,Ken Fujii,Yukihiko Sugita,Takeshi Noda,Hideo Goto,Satoshi Fukuyama,Shinji Watanabe,Gabriele Neumann,Masaaki Oyama,Hiroaki Kitano,Yoshihiro Kawaoka +27 more
TL;DR: A network of virus-host protein interactions based on the stage of the viral life cycle affected upon host factor downregulation is generated and several proteins, notably Golgi-specific brefeldin A-resistant guanine nucleotide exchange factor 1 and JAK1, are validated as potential antiviral drug targets.
Journal ArticleDOI
Initiation of NALT Organogenesis Is Independent of the IL-7R, LTβR, and NIK Signaling Pathways but Requires the Id2 Gene and CD3−CD4+CD45+ Cells
Satoshi Fukuyama,Satoshi Fukuyama,Takachika Hiroi,Yoshifumi Yokota,Paul D. Rennert,Manabu Yanagita,Naotoshi Kinoshita,Seigo Terawaki,Takashi Shikina,Masafumi Yamamoto,Masafumi Yamamoto,Yuichi Kurono,Hiroshi Kiyono,Hiroshi Kiyono,Hiroshi Kiyono +14 more
TL;DR: Evidence is provided that CD3(-)CD4(+)CD45(+) inducer cells can provide an IL-7R-, LTalpha1beta2/LTbetaR-, and NIK-independent tissue organogenesis pathway for secondary lymphoid tissue development.