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Sheila Bingham

Researcher at University of Cambridge

Publications -  520
Citations -  71231

Sheila Bingham is an academic researcher from University of Cambridge. The author has contributed to research in topics: European Prospective Investigation into Cancer and Nutrition & Population. The author has an hindex of 136, co-authored 519 publications receiving 67332 citations. Previous affiliations of Sheila Bingham include International Agency for Research on Cancer & University of East Anglia.

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Blood pressure and interactions between the angiotensin polymorphism AGT M235T and sodium intake: a cross-sectional population study

TL;DR: This large cross-sectional study supports public health recommendations to reduce salt consumption in the population as a whole, and it confirms intervention trial data showing the greatest response to intervention in persons with the AA and TT genotype in the AGT G-6A and M235T polymorphisms.
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Smoking and Lymphoma Risk in the European Prospective Investigation into Cancer and Nutrition

TL;DR: In this prospective study, smoking appeared to increase Hodgkin's lymphoma risk consistently in both genders, whereas B-cell non-Hodgkin'symphoma risk was not associated, and future analysis should involve viral biomarkers and genetic susceptibility markers to elucidate potential mechanisms of smoking-induced carcinogenesis, particularly for Hodgkin't lymphoma.
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Physical health-related quality of life predicts stroke in the EPIC-Norfolk.

TL;DR: Poor physical functional health-related quality of life measured as Short Form-36 predicts subsequent stroke risk independently of known risk factors in a general population.
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Genetic variation in the growth hormone synthesis pathway in relation to circulating insulin-like growth factor-I, insulin-like growth factor binding protein-3, and breast cancer risk: results from the European prospective investigation into cancer and nutrition study.

TL;DR: Common genetic variation in the GH synthesis pathway, as measured by single nucleotide polymorphisms selected in the present study, is not a major determinant of IGF-I and IGFBP-3 circulating levels, and it does not play a major role in altering breast cancer risk.