S
Sverre E. Kjeldsen
Researcher at University of Oslo
Publications - 771
Citations - 95426
Sverre E. Kjeldsen is an academic researcher from University of Oslo. The author has contributed to research in topics: Blood pressure & Left ventricular hypertrophy. The author has an hindex of 94, co-authored 735 publications receiving 89059 citations. Previous affiliations of Sverre E. Kjeldsen include University of Michigan & Cornell University.
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Journal Article
Statin therapy for hypertensive patients
TL;DR: Treatment with 10 mg atorvastatin o.d. in hypertensive patients at moderate risk gives a significant risk reduction of coronary heart disease, independent of baseline level of total cholesterol.
Journal ArticleDOI
Poor reproducibility of masked and white coat uncontrolled hypertension: important new information on MUCH and WUCH.
Sverre E. Kjeldsen,Ingrid Os +1 more
Journal ArticleDOI
Hyper-responders vs. non-responder patients after renal denervation: do they differ?
Alexandre Persu,Michel Azizi,Yu Jin,Sebastian Völz,Ján Rosa,Fadl-Elmula M. Fadl Elmula,Michel Burnier,Patrick B. Mark,Arif Elvan,Jean Renkin,Marc Sapoval,Thomas Kahan,Sverre E. Kjeldsen,Jan A. Staessen +13 more
Journal ArticleDOI
Insulin resistance and sympathetic nervous system activity in hypertensive and normotensive premenopausal women.
TL;DR: It is concluded that in lean, premenopausal hypertensive women insulin sensitivity is not reduced compared with age- and weight-matched normotensive women, but the hypertensives respond to hyperinsulinemia with increased plasma catecholamines, i.e. sympathetic nervous systemic activity.
Book ChapterDOI
Antihypertensive Therapy Benefits: Pleiotropic Versus Blood Pressure-Dependent Mechanisms
TL;DR: A comprehensive meta-analysis of the results of 147 randomized trials involving 464,000 people in the context of epidemiological data from 958,000People indicated that the proportional reduction in cardiovascular disease events is the same or similar regardless of pretreatment blood pressure and the presence of cardiovascular disease.