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Sverre E. Kjeldsen

Researcher at University of Oslo

Publications -  771
Citations -  95426

Sverre E. Kjeldsen is an academic researcher from University of Oslo. The author has contributed to research in topics: Blood pressure & Left ventricular hypertrophy. The author has an hindex of 94, co-authored 735 publications receiving 89059 citations. Previous affiliations of Sverre E. Kjeldsen include University of Michigan & Cornell University.

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Adrenal medullary overactivity in lean, borderline hypertensive young men.

TL;DR: It is suggested that adrenal medullary activation in borderline hypertension mainly characterizes lean subjects, but measures of overweight are independently related to lower plasma E and HR responses.
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Effects of losartan compared with atenolol on lipids in patients with hypertension and left ventricular hypertrophy: the Losartan Intervention For Endpoint reduction in hypertension study.

TL;DR: Losartan blunted the decrease in HDL cholesterol during antihypertensive treatment in the LIFE study, and higher intreatment HDL cholesterol was associated with fewer composite endpoints and may partly explain the better outcome of losartan-based treatment.
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Recent hypertension guidelines: JNC-7 and 2003 ESH/ESC.

TL;DR: The Joint National Committee (JNC) seventh report states that thiazide-type diuretics should be used for drug treatment in most patients with uncomplicated hypertension, either alone or combined with antihypertensive drugs from other classes.
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Treatment of isolated systolic hypertension in diabetes mellitus type 2.

TL;DR: These analyses have clearly demonstrated that blood pressure lowering in ISH confers improved prognosis and reduced cardiovascular and renal outcomes in both diabetics and non‐diabetics.
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Serial assessment of the electrocardiographic strain pattern for prediction of new‐onset heart failure during antihypertensive treatment: the LIFE study

TL;DR: Although the presence of the electrocardiographic (ECG) strain pattern has been associated with an increased risk of developing heart failure, the relationship of regression vs. persistence vs. development of new ECG strain to subsequent HF is unclear.