S
Sverre E. Kjeldsen
Researcher at University of Oslo
Publications - 771
Citations - 95426
Sverre E. Kjeldsen is an academic researcher from University of Oslo. The author has contributed to research in topics: Blood pressure & Left ventricular hypertrophy. The author has an hindex of 94, co-authored 735 publications receiving 89059 citations. Previous affiliations of Sverre E. Kjeldsen include University of Michigan & Cornell University.
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Predictors of 7-year changes in exercise blood pressure : effects of smoking, physical fitness and pulmonary function
TL;DR: Beyond a relatively strong tracking of blood pressures and the expected effect of age, smoking is associated with a 7-year rise in exercise systolic blood pressure whereas relatively higher body mass, physical fitness and pulmonary function are associated with lower exercise blood pressure after 7 years in middle-aged healthy men.
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Hyperresponders vs. nonresponder patients after renal denervation: do they differ?
Alexandre Persu,Michel Azizi,Yu Jin,Sebastian Völz,Ján Rosa,Fadl Elmula M. Fadl Elmula,Antoinette Pechère-Bertschi,Michel Burnier,Patrick B. Mark,Arif Elvan,Jean Renkin,Marc Sapoval,Thomas Kahan,Sverre E. Kjeldsen,Jan A. Staessen +14 more
TL;DR: A major overestimation of BP response after RDN is suggested in extreme responders defined according to office, but not ambulatory BP, which is consistent with previous analysis.
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Assessment of left ventricular function with magnetic resonance imaging vs. echocardiography, contrast echocardiography, and single-photon emission computed tomography in patients with recent ST-elevation myocardial infarction
Nisha Mistry,Sigrun Halvorsen,Pavel Hoffmann,Carl Müller,Ellen Bøhmer,Sverre E. Kjeldsen,Reidar Bjørnerheim +6 more
TL;DR: The data suggest that all four imaging modalities measured EF closely similar after STEMI as demonstrated by a very small bias, and EDV measured by MRI was consistently higher when compared with the other methods which may be caused by different tracing-methods and imaging principles.
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Effect of Angiotensin II Receptor Blockade on Fibrinolysis During Acute Hyperinsulinemia in Patients With Essential Hypertension
TL;DR: The present findings do not preclude more direct effects of angiotensin II or involvement of other receptor subtypes on fibrinolytic variables or the catecholamines, but they cannot demonstrate any effects of 4 weeks of treatment with losartan either in basal conditions or during acute hyperinsulinemia.
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Blood pressure control and components of the metabolic syndrome: the GOOD survey
TL;DR: It is not impaired glucose tolerance which is associated with the poor response to antihypertensive treatment, but visceral obesity and dyslipidemia components of the metabolic syndrome, i.e. hypertriglyceridemia and low HDL cholesterol levels, are associated with resistance to anti Hypertension treatment.