Open Access
Degeneration and regeneration of the nervous system
Santiago Ramón y Cajal
- pp 236-245
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The article was published on 1928-01-01 and is currently open access. It has received 2218 citations till now. The article focuses on the topics: Degeneration (medical) & Regeneration (biology).read more
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Lens-injury-stimulated axonal regeneration throughout the optic pathway of adult rats.
TL;DR: It is suggested, for the first time, that lentogenic stimulation of RGCs is sufficient to induce the formation of growth cones that can override inhibitors at the site of injury, grow through the white matter of the optic nerve, pass through the optic chiasm, and make synaptic connections within the brain.
Journal ArticleDOI
Glial growth factor 2, a soluble neuregulin, directly increases Schwann cell motility and indirectly promotes neurite outgrowth.
TL;DR: GGF2 concentrations greater than necessary to saturate the mitotic response induce the secretion by Schwann cells of activities that promote sympathetic neuron survival and outgrowth, which support a model in which neuregulins secreted by peripheral neurons are key components of reciprocal neuron–glia interactions that are important for peripheral nerve development and regeneration.
Journal ArticleDOI
Enhancement of peripheral nerve regeneration.
TL;DR: The biology of the regeneration response of the nerve to injury is reviewed and many of the factors that enhance nerve growth are discussed that support and enhance nerve regeneration after injury.
Journal ArticleDOI
Cell-to-cell transfer of glial proteins to the squid giant axon: The glia- neuron protein transfer hypothesis
TL;DR: It is concluded that the adaxonal glial cells synthesize a class of proteins which are transferred to the giant axon, which is supplemental to that provided by axonal transport from the cell body.
Journal ArticleDOI
Environmental influences on brain neurotrophins in rats.
TL;DR: Data from the laboratory have revealed that enriched environment increased nerve growth factor gene expression and protein levels in the hippocampus, and this may contribute to events underlying environmentally induced neural plasticity.
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