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Journal ArticleDOI

Nociception from skeletal muscle in relation to clinical muscle pain.

Siegfried Mense
- 01 Sep 1993 - 
- Vol. 54, Iss: 3, pp 241-289
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This article is published in Pain.The article was published on 1993-09-01. It has received 816 citations till now. The article focuses on the topics: Skeletal muscle & Nociception.

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Citations
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The induction of pain: an integrative review

TL;DR: A global account of mechanisms involved in the induction of pain is provided, including neuronal pathways for the transmission of nociceptive information from peripheral nerve terminals to the dorsal horn, and therefrom to higher centres.
Journal ArticleDOI

Acute and Chronic Craniofacial Pain: Brainstem Mechanisms of Nociceptive Transmission and Neuroplasticity, and Their Clinical Correlates

TL;DR: The recent findings provide new insights into craniofacial pain behavior and are particularly relevant to many approaches currently in use for the management of pain and to the development of new diagnostic and therapeutic procedures aimed at manipulating peripheral inputs and central processes underlying nociceptive transmission and its control within the VBSNC.
Journal ArticleDOI

Trunk muscle activation in low-back pain patients, an analysis of the literature

TL;DR: It is proposed that motor control changes in patients are functional in that they enhance spinal stability and suggest an alternative model to explain the alterations of trunk muscle recruitment due to low-back pain.
Journal ArticleDOI

Multi-functional dielectric elastomer artificial muscles for soft and smart machines

TL;DR: The octopus arm is an example of a soft actuator with a virtually infinite number of degrees of freedom (DOF) as discussed by the authors, which utilizes neural ganglia to process sensory data at the local “arm” level and perform complex tasks.
References
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Journal ArticleDOI

Leukotrienes: mediators of immediate hypersensitivity reactions and inflammation

TL;DR: Leukotrienes C4, D4, and E4, which are released from the lung tissue of asthmatic subjects exposed to specific allergens, seem to play a pathophysiological role in immediate hypersensitivity reactions.
Journal ArticleDOI

Glutamate, nitric oxide and cell-cell signalling in the nervous system

TL;DR: In this paper, it was shown that NO is produced enzymatically in postsynaptic structures in response to activation of excitatory amino acid receptors and diffuses out to act on neighbouring cellular elements, probably presynaptic nerve endings and astrocyte processes.
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