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Fernando Almeida

Researcher at Pontifícia Universidade Católica de São Paulo

Publications -  90
Citations -  3261

Fernando Almeida is an academic researcher from Pontifícia Universidade Católica de São Paulo. The author has contributed to research in topics: Medicine & Poison control. The author has an hindex of 17, co-authored 81 publications receiving 2718 citations. Previous affiliations of Fernando Almeida include Faculdade de Engenharia da Universidade do Porto & Cornell University.

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Physiological role of silent receptors of atrial natriuretic factor

TL;DR: The results show that the majority of the renal receptors of ANF are biologically silent, and this new class of receptors may serve as specific peripheral storage-clearance binding sites, acting as a hormonal buffer system to modulate plasma levels of ANf.
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Mycophenolate Mofetil versus Cyclophosphamide for Induction Treatment of Lupus Nephritis

Gerald B. Appel, +98 more
TL;DR: Although most patients in both treatment groups experienced clinical improvement, the study did not meet its primary objective of showing that MMF was superior to IVC as induction treatment for lupus nephritis.
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Diretrizes Brasileiras de Hipertensão Arterial – 2020

Weimar Kunz Sebba Barroso, +97 more
TL;DR: The Brazilian Guidelines of Hypertension -2020 as mentioned in this paper have been published for the first time in the year 2020, and are based on the definition, epidemiology, and primary prevention.
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Atrial natriuretic factor increases hematocrit and decreases plasma volume in nephrectomized rats.

TL;DR: This effect explains, at least in part, the ANF-induced decrease in cardiac output and blood pressure in normotensive animals and may play an important role in the homeostatic regulation of plasma volume.
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Clearance function of type C receptors of atrial natriuretic factor in rats

TL;DR: The data demonstrate that C-ANF receptors are mainly responsible for the very large volume of distribution and fast MCR of ANF in the rat, and are likely to play an important role in the homeostasis of circulating ANF.