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Giovanni B. Frisoni

Researcher at University of Geneva

Publications -  956
Citations -  55940

Giovanni B. Frisoni is an academic researcher from University of Geneva. The author has contributed to research in topics: Dementia & Medicine. The author has an hindex of 101, co-authored 871 publications receiving 46199 citations. Previous affiliations of Giovanni B. Frisoni include Geneva College & University of Rome Tor Vergata.

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How fast will it go, doc? New tools for an old question from patients with Alzheimer disease

TL;DR: A number of clinical and genetic features have been found in early studies to be associated with faster disease progression, such as parkinsonism and psychosis,1 aphasia,2 and apoE genotype as mentioned in this paper.
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Depression and somatic symptoms in the elderly: the role of cognitive function

TL;DR: In this article, the authors developed a study on the quality of life of elderly people and involved 462 community-dwelling subjects aged 75 and over living in the city centre of Brescia and Padua, northern Italy.
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Co-occurrence of disadvantage conditions in elderly subjects with depressive symptoms.

TL;DR: The most disadvantaged tertile of various conditions (age, social support, cognition, social interactions, self evaluation of health, disability, number of diseases, and somatic symptoms) was associated with greater risk of symptomatic depression as mentioned in this paper.

Research report Co-occurrence of disadvantage conditions in elderly subjects with depressive symptoms

TL;DR: The most disadvantaged tertile of various conditions (age, social support, cognition, social interactions, self evaluation of health, disability, number of diseases, and somatic symptoms) was associated with greater risk of symptomatic depression.
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Association of Blood Pressure and Genetic Background With White Matter Lesions in Patients With Mild Cognitive Impairment

TL;DR: BP and gene putative risk factors for cerebrovascular disease are differentially associated with WMLs in two MCI groups of different WML severity, which might develop for the convergence of innate with acquired factors.