G
Giovanni B. Frisoni
Researcher at University of Geneva
Publications - 956
Citations - 55940
Giovanni B. Frisoni is an academic researcher from University of Geneva. The author has contributed to research in topics: Dementia & Medicine. The author has an hindex of 101, co-authored 871 publications receiving 46199 citations. Previous affiliations of Giovanni B. Frisoni include Geneva College & University of Rome Tor Vergata.
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Journal ArticleDOI
Occipital sources of resting-state alpha rhythms are related to local gray matter density in subjects with amnesic mild cognitive impairment and Alzheimer's disease
Claudio Babiloni,Claudio Del Percio,Marina Boccardi,Roberta Lizio,Susanna Lopez,Filippo Carducci,Nicola Marzano,Andrea Soricelli,Raffaele Ferri,Antonio Ivano Triggiani,Annapaola Prestia,Serenella Salinari,Paul E. Rasser,Erol Başar,Francesco Famà,Flavio Nobili,Görsev Yener,Derya Durusu Emek-Savaş,Derya Durusu Emek-Savaş,Loreto Gesualdo,Ciro Mundi,Paul M. Thompson,Paolo Maria Rossini,Giovanni B. Frisoni +23 more
TL;DR: The results suggest that the amplitude of occipital sources of resting-state alpha rhythms is related to AD neurodegeneration in Occipital lobe along pathologic aging.
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Alexithymia in healthy women: A brain morphology study
Genoveffa Borsci,Marina Boccardi,Roberta Rossi,Giuseppe Rossi,Jorge Perez,Matteo Bonetti,Giovanni B. Frisoni +6 more
TL;DR: The findings suggest that the ability to process emotional aspects of the self correlates with morphology of a specific set of cerebral structures known to be involved in decision making and self awareness and rich in neurons subserving social competence.
Journal ArticleDOI
Rare mutations in SQSTM1 modify susceptibility to frontotemporal lobar degeneration.
Julie van der Zee,Tim Van Langenhove,Gabor G. Kovacs,Lubina Dillen,William Deschamps,Sebastiaan Engelborghs,Radoslav Matěj,Mathieu Vandenbulcke,Anne Sieben,Bart Dermaut,Katrien Smets,Philip Van Damme,Céline Merlin,Annelies Laureys,Marleen Van den Broeck,Maria Mattheijssens,Karin Peeters,Luisa Benussi,Giuliano Binetti,Roberta Ghidoni,Barbara Borroni,Alessandro Padovani,Silvana Archetti,Pau Pastor,Pau Pastor,Cristina Razquin,Sara Ortega-Cubero,Sara Ortega-Cubero,Isabel Hernández,Mercè Boada,Agustín Ruiz,Alexandre de Mendonça,Gabriel Miltenberger-Miltenyi,Frederico Simões do Couto,Sandro Sorbi,Benedetta Nacmias,Silvia Bagnoli,Caroline Graff,Caroline Graff,Huei Hsin Chiang,Huei Hsin Chiang,Håkan Thonberg,Robert Perneczky,Robert Perneczky,Janine Diehl-Schmid,Panagiotis Alexopoulos,Giovanni B. Frisoni,Christian Bonvicini,Matthis Synofzik,Walter Maetzler,Jennifer Müller vom Hagen,Ludger Schöls,Tobias B. Haack,Tim M. Strom,Holger Prokisch,Oriol Dols-Icardo,Jordi Clarimón,Alberto Lleó,Isabel Santana,Maria Rosário Almeida,Beatriz Santiago,Michael T. Heneka,Frank Jessen,Alfredo Ramirez,Raquel Sánchez-Valle,Albert Lladó,Ellen Gelpi,Stayko Sarafov,Ivailo Tournev,Ivailo Tournev,Albena Jordanova,Eva Parobkova,Gian Maria Fabrizi,Silvia Testi,Eric Salmon,Thomas Ströbel,Patrick Santens,Wim Robberecht,Peter De Jonghe,Jean Jacques Martin,Patrick Cras,Rik Vandenberghe,Peter Paul De Deyn,Peter Paul De Deyn,Marc Cruts,Kristel Sleegers,Christine Van Broeckhoven +86 more
TL;DR: Investigation of the SQSTM1 coding sequence for mutations in an extended cohort of 1,808 patients with frontotemporal lobar degeneration showed that, comparable to other FTLD/ALS genes, SQ STM1 mutations are associated with TDP-43 pathology.
Journal ArticleDOI
Cortical sources of resting state electroencephalographic rhythms in Parkinson’s disease related dementia and Alzheimer’s disease
Claudio Babiloni,Maria Francesca De Pandis,Fabrizio Vecchio,Paola Buffo,Fabiola Sorpresi,Giovanni B. Frisoni,Paolo Maria Rossini +6 more
TL;DR: The results suggest that topography and frequency of eyes-closed resting state cortical EEG rhythms distinguished PDD and AD groups and report the existence of different effects of neurodegeneration on the cortical neural synchronization mechanisms generating resting state EEG rhythms in PDd and AD patients.
Journal ArticleDOI
In vivo mapping of amyloid toxicity in Alzheimer disease.
TL;DR: The medial temporal lobe might be highly susceptible to amyloid toxicity, whereas neocortical areas might be more resilient.