J
Jane Morley Kotchen
Researcher at Medical College of Wisconsin
Publications - 96
Citations - 32980
Jane Morley Kotchen is an academic researcher from Medical College of Wisconsin. The author has contributed to research in topics: Blood pressure & Women's Health Initiative. The author has an hindex of 43, co-authored 96 publications receiving 31310 citations. Previous affiliations of Jane Morley Kotchen include National Institutes of Health & University of Wisconsin–Milwaukee.
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Journal ArticleDOI
Arterial Pressure, Left Ventricular Mass, and Aldosterone in Essential Hypertension
Areeg El-Gharbawy,Vishwanatha S. Nadig,Jane Morley Kotchen,Clarence E. Grim,Kiran B. Sagar,Mary L. Kaldunski,Pavel Hamet,Zdenka Pausova,Daniel Gaudet,Francis Gossard,Theodore A. Kotchen +10 more
TL;DR: Results are consistent with the hypothesis that aldosterone contributes to elevated arterial pressure in obese black American and obese white French Canadian patients with essential hypertension and to the attenuated nocturnal decline of blood pressure and left ventricular hypertrophy in obese, hypertensive black Americans.
Journal ArticleDOI
Depressive symptoms and incidence of mild cognitive impairment and probable dementia in elderly women: the Women's Health Initiative Memory Study.
Joseph S. Goveas,Mark A. Espeland,Nancy Fugate Woods,Sylvia Wassertheil-Smoller,Jane Morley Kotchen +4 more
TL;DR: In this paper, the authors examined whether significant depressive symptoms in postmenopausal women increase the risk of subsequent mild cognitive impairment (MCI) and dementia, and concluded that women without depression who endorsed a remote history of depression had a higher risk of developing dementia.
Journal ArticleDOI
Genetic determinants of hypertension: identification of candidate phenotypes.
Theodore A. Kotchen,Jane Morley Kotchen,Clarence E. Grim,Varghese George,Mary L. Kaldunski,Allen W. Cowley,Pavel Hamet,Thomas Chelius +7 more
TL;DR: Evidence for the heritability of hypertension-related phenotypes in hypertensive, hyperlipidemic black sib pairs is described to facilitate the identification of specific genetic determinants of hypertension in blacks withhyperlipidemia.
Journal ArticleDOI
Hyperaldosteronism and Hypertension: Ethnic Differences
Clarence E. Grim,Allen W. Cowley,Pavel Hamet,Daniel Gaudet,Mary L. Kaldunski,Jane Morley Kotchen,Shanthi Krishnaswami,Zdenka Pausova,Richard J. Roman,Johanne Tremblay,Theodore A. Kotchen +10 more
TL;DR: Correlations of blood pressure with aldosterone were more consistent and more striking in blacks than in French Canadians, and these observations are consistent with the hypothesis that ald testosterone-induced volume expansion is an important contributor to hypertension, especially in blacks.
Journal ArticleDOI
Arthritis Increases the Risk for Fractures---Results from the Women’s Health Initiative
Nicole C. Wright,Jeffrey R. Lisse,Brian Walitt,Charles B. Eaton,Zhao Chen,Elizabeth G. Nabel,Jacques E. Rossouw,Shari E. Ludlam,Linda M. Pottern,Joan McGowan,Leslie G. Ford,Nancy L. Geller,Ross L. Prentice,Garnet L. Anderson,Andrea Z. LaCroix,Charles Kooperberg,Ruth E. Patterson,Anne McTiernan,Sally A. Shumaker,Evan A. Stein,Steven R. Cummings,Sylvia Wassertheil-Smoller,Aleksandar Rajkovic,JoAnn E. Manson,Annlouise R. Assaf,Lawrence S. Phillips,Shirley A.A. Beresford,Judith Hsia,Rowan T. Chlebowski,Evelyn P Whitlock,Bette J. Caan,Jane Morley Kotchen,Barbara V. Howard,Linda Van Horn,Henry R. Black,Marcia L. Stefanick,Dorothy S. Lane,Rebecca D. Jackson,Cora E. Lewis,Tamsen Bassford,Jean Wactawski-Wende,John A Robbins,F. Allan Hubbell,Lauren Nathan,Robert Langer,Margery Gass,Marian C. Limacher,David Curb,Robert B. Wallace,Judith K. Ockene,Norman L. Lasser,Mary Jo O'Sullivan,Karen L. Margolis,Robert L. Brunner,Gerardo Heiss,Lewis H. Kuller,Karen C. Johnson,Robert G. Brzyski,Gloria E. Sarto,Mara Z. Vitolins,Susan L. Hendrix +60 more
TL;DR: The increase in fracture risk confirms the importance of fracture prevention in patients with RA and OA, and report of arthritis was associated with increased risk for spine, hip, and any clinical fractures.