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Melissa J. Alldred

Researcher at Nathan Kline Institute for Psychiatric Research

Publications -  45
Citations -  2904

Melissa J. Alldred is an academic researcher from Nathan Kline Institute for Psychiatric Research. The author has contributed to research in topics: Basal forebrain & Cholinergic neuron. The author has an hindex of 24, co-authored 43 publications receiving 2365 citations. Previous affiliations of Melissa J. Alldred include Pennsylvania State University & New York University.

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Microarray Analysis of Hippocampal CA1 Neurons Implicates Early Endosomal Dysfunction During Alzheimer's Disease Progression

TL;DR: The hypothesis that neuronal endosomal dysfunction is associated with preclinical AD is supported and increased endocytic pathway activity, driven by elevated rab GTPase expression, may result in long-term deficits in hippocampal neurotrophic signaling and represent a key pathogenic mechanism underlying AD progression.
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The γ2 Subunit of GABAA Receptors Is a Substrate for Palmitoylation by GODZ

TL;DR: GODZ-mediated palmitoylation represents a novel posttranslational modification that is selective forγ subunit-containing GABAA receptor subtypes, a mechanism that is likely to be important for regulated trafficking of these receptors in the secretory pathway.
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Autophagy flux in CA1 neurons of Alzheimer hippocampus: Increased induction overburdens failing lysosomes to propel neuritic dystrophy.

TL;DR: It is proposed that sustained induction of autophagy in the face of progressively declining lysosomal clearance of substrates explains the uncommonly robust autophagic pathology and neuritic dystrophy implicated in AD pathogenesis.
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Distinct γ2 Subunit Domains Mediate Clustering and Synaptic Function of Postsynaptic GABAA Receptors and Gephyrin

TL;DR: A novel mechanism involved in targeting of GABAA receptors and gephyrin to inhibitory synapses is pointed to in γ2 subunit mutant neurons.