N
Nicola L. Harris
Researcher at Monash University
Publications - 121
Citations - 10360
Nicola L. Harris is an academic researcher from Monash University. The author has contributed to research in topics: Immune system & Heligmosomoides polygyrus. The author has an hindex of 40, co-authored 110 publications receiving 8780 citations. Previous affiliations of Nicola L. Harris include University of Lausanne & University of Otago.
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Bystander suppression of allergic airway inflammation by lung resident memory CD8+ T cells
TL;DR: Data indicate that effector/memory CD8+ T cells present in the airways produce IFN-gamma after inflammatory stimuli, independent of specific-antigen, and as a consequence play a key role in modifying the degree and frequency of allergic responses in the lung.
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The functions of mucosal T cells in containing the indigenous commensal flora of the intestine.
TL;DR: The roles of intestinal T cells in initiating and regulating innate and adaptive mucosal immune responses of the mucosalimmune system that avoid or limit penetration of the commensal intestinal bacteria are discussed.
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Helminth–Bacterial Interactions: Cause and Consequence
TL;DR: Intestinal helminths, along with mutualistic microbes, have cohabited the intestine of mammals throughout evolution and may favor the establishment of a particular microbiome with either positive or negative consequences for the overall health and well-being of the host.
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Intestinal bacteria induce TSLP to promote mutualistic T-cell responses
Ilaria Mosconi,Markus B. Geuking,Mario M. Zaiss,Joanna Massacand,C Aschwanden,C K C Kwong Chung,Kathy D. McCoy,Nicola L. Harris +7 more
TL;DR: An important role for TSLP-TSLPR signaling is revealed in promoting steady-state mutualistic T-cell responses following intestinal bacterial colonization and limiting the expansion of colonic T helper type 17 cells.
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Links Between Inflammatory Bowel Disease and Chronic Obstructive Pulmonary Disease.
TL;DR: This review will focus on the gut-lung axis in the context of IBD and COPD, highlighting the role of environmental and genetic factors and the impact of microbial dysbiosis on chronic inflammation in the intestinal tract and lung.