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Noriyuki Koibuchi

Researcher at Gunma University

Publications -  204
Citations -  5876

Noriyuki Koibuchi is an academic researcher from Gunma University. The author has contributed to research in topics: Thyroid hormone receptor & Cerebellum. The author has an hindex of 38, co-authored 191 publications receiving 5396 citations. Previous affiliations of Noriyuki Koibuchi include Brigham and Women's Hospital & Dokkyo University.

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Journal ArticleDOI

TRAM-1, A Novel 160-kDa Thyroid Hormone Receptor Activator Molecule, Exhibits Distinct Properties from Steroid Receptor Coactivator-1 *

TL;DR: The results suggest that TRAM-1 is a coactivator that may exhibit its activity by interacting with subdomains of NRs other than the AF-2 region, in contrast to SRC-1/TIF2.
Journal ArticleDOI

Thyroid Hormone Action and Brain Development

TL;DR: Developing rodent cerebellum might be an excellent model for studying the molecular mechanisms of TH action in the brain because perinatal hypothyroidism greatly affects its ontogeny.
Journal ArticleDOI

Consensus Paper: Cerebellar Development.

TL;DR: The main processes of cerebellar ontogenesis are described, highlighting the neurogenic strategies used by developing progenitors, the genetic programs involved in cell fate specification, the progressive changes of structural organization, and some of the better-known abnormalities associated with developmental disorders of the cerebellum.
Reference BookDOI

Handbook of the cerebellum and cerebellar disorders

TL;DR: The cerebellar primordium develops dorsally at an intermediate anteroposterior level of the neural tube and contributes to a large number of nuclei in the isthmic region.
Journal ArticleDOI

Developmental thyroid hormone disruption: prevalence, environmental contaminants and neurodevelopmental consequences.

TL;DR: Empirical, preclinical and animal research has clearly identified the critical role of TH in brain development and additional work is required to understand the impact of low level perturbations of the thyroid axis to evaluate the risk associated with environmental contaminants with thyroid action.