Sarrah Ben M’Barek

TL;DR: The genome of M. graminicola was sequenced completely and found that it contained very few genes for enzymes that break down plant cell walls, which was more similar to endophytes than to pathogens, which may have evolved from endophytic ancestors.

TL;DR: Evidence is given that HGT of the ToxA gene from Stagonospora nodorum to Pyrenophora tritici-repentis enabled the latter fungus to cause a serious disease in wheat, and the mechanisms HGT and HCT and their impact on potential emergence of fungal plant pathogens adapted to new host plants will be discussed.

TL;DR: High genome plasticity could be among the strategies enabling this versatile pathogen to quickly overcome adverse biotic and abiotic conditions in wheat fields.

TL;DR: It is shown that these treatments allow sex and modulate parenthood in the fungal wheat pathogen Zymoseptoria tritici, and demonstrates that neither host resistance nor fungicide treatment suppresses fungal sexual reproduction, thus unveiling implications of fungal sex in plant disease control.

TL;DR: Results reveal that generally adopted criteria, such as protein size, cysteine residues and expression during pathogenesis, may preclude an unbiased effector discovery and genetic mapping of genomic regions involved in specificity render alternative effector candidates that do not match the aforementioned criteria, but should nevertheless be considered as promising new leads for effectors that are crucial for the Z. tritici-wheat pathosystem.