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Wilburn E. Reddick

Researcher at St. Jude Children's Research Hospital

Publications -  169
Citations -  9746

Wilburn E. Reddick is an academic researcher from St. Jude Children's Research Hospital. The author has contributed to research in topics: Neurocognitive & White matter. The author has an hindex of 52, co-authored 164 publications receiving 8709 citations. Previous affiliations of Wilburn E. Reddick include University of Tennessee Health Science Center & Baylor College of Medicine.

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The challenge of mapping the human connectome based on diffusion tractography

Klaus H. Maier-Hein, +76 more
TL;DR: The encouraging finding that most state-of-the-art algorithms produce tractograms containing 90% of the ground truth bundles (to at least some extent) is reported, however, the same tractograms contain many more invalid than valid bundles, and half of these invalid bundles occur systematically across research groups.
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Late neurocognitive sequelae in survivors of brain tumours in childhood.

TL;DR: Differences in cognitive development have been described most thoroughly among children treated for posterior-fossa tumours, specifically medulloblastomas and ependymomas, which account for about 30% of all newly diagnosed cases of brain tumours in children.
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Patterns of intellectual development among survivors of pediatric medulloblastoma: a longitudinal analysis.

TL;DR: The hypothesis that MB patients demonstrate a decline in IQ values because of an inability to acquire new skills and information at a rate comparable to their healthy same-age peers, as opposed to a loss of previously acquired information and skills is supported.
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Automated segmentation and classification of multispectral magnetic resonance images of brain using artificial neural networks

TL;DR: This fully automated technique produces reliable and reproducible MR image segmentation and classification while eliminating intra- and interobserver variability.
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Risks of Young Age for Selected Neurocognitive Deficits in Medulloblastoma Are Associated With White Matter Loss

TL;DR: The present results suggest that, at least for some cognitive functions, deficient development and/or loss of NAWM after CRT may provide a neuroanatomical substrate for the adverse impact of a young age at the time of CRT.