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Andreas Strasser
Researcher at Walter and Eliza Hall Institute of Medical Research
Publications - 537
Citations - 75592
Andreas Strasser is an academic researcher from Walter and Eliza Hall Institute of Medical Research. The author has contributed to research in topics: Apoptosis & Programmed cell death. The author has an hindex of 128, co-authored 509 publications receiving 66903 citations. Previous affiliations of Andreas Strasser include University of Alabama at Birmingham & Basel Institute for Immunology.
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Journal ArticleDOI
Lymphocyte Survival--Ignorance Is BLys
Yacine Laâbi,Andreas Strasser +1 more
TL;DR: The receptors BCMA and TACI and their ligands BAFF/BLys and APRIL, respectively, are important for B lymphocyte survival, proliferation, and differentiation.
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Viewing BCL2 and cell death control from an evolutionary perspective.
TL;DR: It is proposed that a cell suicide process that initially evolved as a mechanism for defense against intracellular parasites was then also used in multicellular organisms for morphogenesis and to maintain the correct number of cells in adults by balancing cell production by mitosis.
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Puma indirectly activates Bax to cause apoptosis in the absence of Bid or Bim
Anissa Jabbour,Jacki E Heraud,Carmel Patricia Daunt,Thomas Kaufmann,Thomas Kaufmann,Jarrod J. Sandow,Lorraine A. O'Reilly,Bernard A. Callus,Angel F. Lopez,Andreas Strasser,David L. Vaux,Paul G Ekert +11 more
TL;DR: Data indicate that Puma functions, in the context of induced overexpression or IL-3 deprivation, primarily by binding and inactivating anti-apoptotic Bcl-2 family members.
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One synchronous wave of B cell development in mouse fetal liver changes at day 16 of gestation from dependence to independence of a stromal cell environment.
TL;DR: Results indicate that B cell development occurs in one wave with synchronous steps of changes from a mitogen-insensitive, sIg- , stromal cell dependent to aMitogen-reactive, s Ig+, stromAL cell- independent B lineage line.
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Normal Thymocyte Negative Selection in TRAIL-deficient Mice
Erika Cretney,Adam P Uldrich,Stuart P. Berzins,Andreas Strasser,Dale I. Godfrey,Mark J. Smyth +5 more
TL;DR: Investigation of intrathymic negative selection of TRAIL-deficient thymocytes, using four well-established models, was unable to demonstrate a role for TRAIL signaling in any of these models, suggesting that this pathway is not a critical factor for thymocyte negative selection.