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Edward Giovannucci

Researcher at Harvard University

Publications -  1819
Citations -  202335

Edward Giovannucci is an academic researcher from Harvard University. The author has contributed to research in topics: Cancer & Prostate cancer. The author has an hindex of 206, co-authored 1671 publications receiving 179875 citations. Previous affiliations of Edward Giovannucci include University of California, San Francisco & American Cancer Society.

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Prospective study of bowel movement, laxative use, and risk of colorectal cancer among women.

TL;DR: Findings do not support an association between infrequent bowel movement, laxative use, and risk of colorectal cancer and indicate that simple questions directed at bowel movement frequency are unlikely to enhance the ability to predict coloreCTal cancer risk.
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Prospective study of alcohol consumption patterns in relation to symptomatic gallstone disease in men.

TL;DR: It is suggested that frequent, moderate intake of alcohol decreases the risk for symptomatic gallstone disease, in contrast to infrequent or episodic alcohol intake.
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Meat intake and risk of diverticulitis among men

TL;DR: Red meat intake, particularly unprocessed red meat, was associated with an increased risk of Diverticulitis, and the findings provide practical dietary guidance for patients at risk of diverticULitis.
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Ejaculation Frequency and Risk of Prostate Cancer: Updated Results with an Additional Decade of Follow-up

TL;DR: Ejaculation frequency at age 40-49 yr was positively associated with age-standardized body mass index, physical activity, divorce, history of sexually transmitted infections, and consumption of total calories and alcohol, and associations were driven by low-risk disease.
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Prospective Analysis of Body Mass Index, Physical Activity, and Colorectal Cancer Risk Associated with β-Catenin (CTNNB1) Status

TL;DR: It is argued that obesity and physical inactivity are associated with a higher risk of CTNNB1-negative colorectal cancer but not with CTNNb1-positive cancer risk, and energy balance and metabolism status exerts its effect in a specific carcinogenesis pathway that is less likely dependent on WNT/CTNNB 1 activation.