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Eliane Gluckman

Researcher at University of Paris

Publications -  694
Citations -  48573

Eliane Gluckman is an academic researcher from University of Paris. The author has contributed to research in topics: Transplantation & Bone marrow. The author has an hindex of 112, co-authored 679 publications receiving 46415 citations. Previous affiliations of Eliane Gluckman include Medical College of Wisconsin & Leiden University.

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Palivizumab Treatment of Respiratory Syncytial Virus Infection after Allogeneic Hematopoietic Stem Cell Transplantation

TL;DR: Among 40 allogeneic stem cell transplant recipients who developed symptomatic respiratory syncytial virus infection, including 22 patients with lower respiratory tract infection, 19 received palivizumab and did not prevent progression to lower respiratory infection and had no impact on the overall survival rate.
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Outcomes after related and unrelated umbilical cord blood transplantation for hereditary bone marrow failure syndromes other than Fanconi anemia.

TL;DR: In patients with hereditary bone marrow failure syndromes, related umbilical cord blood transplantation is associated with excellent outcomes while increasing cell dose and better HLA matching might provide better results in unrelated umbilicals cord blood transplants.
Journal Article

Treatment with marrow transplantation or immunosuppression of childhood acquired severe aplastic anemia: a report from the EBMT SAA Working Party.

TL;DR: The results suggest that IS is a satisfactory alternative therapy for children with moderately SAA in the absence of an HLA-identical sibling, although BMT remains the treatment of choice.
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Contribution of growth hormone deficiency to the growth failure that follows bone marrow transplantation

TL;DR: The individual height changes between BMT and the last clinical evaluation before GH therapy were not correlated with the age at BMT, GH peak after stimulation, plasma IGF-I concentration, or IGFBP-3 concentration, which suggest that bone irradiation induces lesions causing resistance to IGF-i.